Cardiomyopathy

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Related to ARVC: Arrhythmogenic right ventricular cardiomyopathy, ARVD

cardiomyopathy

[¦kärd·ē·ō‚mī′äp·ə·thē]
(medicine)

Cardiomyopathy

 

(myocardosis), a term often applied to a broad group of heart diseases; specifically, noninflammatory lesions of the heart muscle (myocardium) resulting from a disturbance in myocardial metabolism. Among the causes of cardiomyopathy are nutritional disorders (alimentary dystrophy and avitaminosis, for example); protein metabolism disorders in hepatic or renal insufficiency and podagra; disturbances of carbohydrate metabolism (diabetes mellitus) and electrolyte metabolism; and endocrine disorders such as those associated with thyrotoxicosis and with hypoxia in impairment of coronary circulation, anemia, and mountain sickness. Myocardosis may also be caused by overstraining the myocardium and by exogenous poisons, such as carbon monoxide and alcohol.

In many cases the patient has no specific symptoms in the early stages; however, there may be shortness of breath and disagreeable sensations in the heart. Cardiomyopathy is manifested by dull, distant heart sounds, electrocardiographic changes, systolic murmur, extrasystole, and, more rarely, other types of arrhythmia. Severe cardiomyopathy weakens the heart contractions and may cause cardiac insufficiency. The changes associated with cardiomyopathy are usually reversible and disappear with the elimination of the underlying disease.

The cure includes treatment of the underlying disease and administration of agents that improve metabolic processes in the myocardium.

REFERENCE

Kedrov, A. A. Bolezni myshtsy serdtsa. Leningrad, 1963.

N. R. PALEEV

References in periodicals archive ?
Symptoms of ARVC include palpitations, light-headedness, fainting, breathlessness, abnormal heat rhythms, swollen ankles or legs, swelling in the abdomen, risk of sudden death on exertion.
ARVC is not curable, but most of the symptoms caused by the condition can be controlled using medicines.
Treatment of ARVC has remained disappointing, as the only curative option is orthotopic cardiac transplantation, which is considered only in individuals with progressive heart failure or intractable ventricular arrhythmia.
The 2006 guidelines note that fibrofatty changes in the right ventricle of ARVC patients may interfere with adequate lead positioning and arrhythmia sensing (11).
Several theories of the origin and pathogenesis of ARVC have been advanced.
It is now recognized that Uhl anomaly and ARVC are 2 separate entities.
The diagnosis was ARVC and an implantable cardioverter/defibrillator was placed for life-threatening sustained ventricular tachycardia.
The diagnosis of ARVC is based on the presence of familial inheritance, the structural and functional pathologies of right ventricle, arrhythmias, depolarization and repolarization abnormalities on ECG.
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