The Babinski sign
provides evidence of a PTD, but sometimes either its absence or its doubtable presence make further clinical and paraclinical examinations necessary in a patient under suspicion of a PTD, simply because its sensitivity is low.
Subsequently, Cook et al (6) examined the reliability and sensitivity of 7 clinical tests: Babinski sign
, clonus, Hoffmann sign, inverted supinator sign, hand withdrawal reflex, suprapatellar quadriceps reflex, and upper extremity deep tendon reflexes.
The only abnormalities found on physical examination of the adults were a positive Babinski sign
and absent rotular reflexes in 3 of the 23 siblings' mothers.
We also conducted a search through medical journals that examined the Babinski sign
and its history.
Our previous experimental and clinical observations indicate that a positive ice-water test represents a release from central inhibition, analogous to the release of the Babinski sign
after pyramidal tract lesions |12, 18, 19~.
Neurologic examination showed left homonymous hemianopia, increased deep tendon reflexes in the left hemibody, and the Babinski sign
on the left hallux.
The deep tendon reflexes were +++/4 and there was bilateral Babinski sign
Relationship of periodic movements in sleep (nocturnal myoclonus) and the Babinski sign
The Babinski sign
was absent on the left and neutral on the right.
There was no Babinski sign
on the left side, and left cerebral tests spoil.
Neurologic deficit was localized to the left side and revealed a homonymous hemianopia, facial weakness, deviation of the tongue to the left, flaccid hemiparesis, hypoesthesis to pinprick sensation, hyperreflexia, and Babinski sign
Results of a sensory examination were normal, and patient's reflexes were largely intact; Babinski sign