dichloroacetic acid


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dichloroacetic acid

[dī¦klȯr·ō·ə¦sēd·ik ′as·əd]
(organic chemistry)
CHCl2COOH A strong liquid acid, formed by chlorinating acetic acid; used in organic synthesis.
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Photocatalytic intrinsic reaction kinetics I: Mineralization of dichloroacetic acid.
2] Nanocomposites as Highly Active Photocatalysts for the Photooxidation of Dichloroacetic Acid, Journal of Physical Chemistry C: 115(13), 5784-5791 (2011).
Dichloroacetic acid (DCA) and trichloroacetic acid (TCA), the HAA5s most commonly found, are also of concern.
Liquid chromatography electrospray ionization tandem mass spectrometry analysis method for simultaneous detection of trichloroacetic acid, dichloroacetic acid, S-(1,2-dichlorovinyl)glutathione and S-(1,2-dichlorovinyl)-L-cysteine.
Adesina, Oxidative Photomineralization of Dichloroacetic Acid in an Externally-Irradiated Rectangular Bubble Tank Reactor: Computational Fluid Dynamics Modeling and Experimental Verification Studies, Industrial & Engineering Chemistry Research: 49 (15), 6722-6734 (2010).
Conversion of trichloroacetic acid to dichloroacetic acid in biological samples.
Hepatocarcinogenicity in the male B6C3F1 mouse following a lifetime exposure to dichloroacetic acid in the drinking water: dose-response determination and modes of action.
Moreover, there are a number of metabolites of potential toxicologic interest, such as chloral, dichloroacetic acid, and those derived from glutathione conjugation, for which reliable pharmacokinetic data is sparse because of analytical difficulties or low concentrations in systemic circulation.
Studies of the TCE metabolites dichloroacetic acid (DCA), trichloroacetic acid (TCA), and chloral hydrate suggest that both DCA and TCA are involved in TCE-induced liver tumorigenesis and that many DCA effects are consistent with conditions that increase the risk of liver cancer in humans.
Chlorination of water supplies generates several TCE metabolites, including chloral hydrate, trichloroacetic acid (TCA), and dichloroacetic acid (Miller and Uden 1983).
Our findings suggest a critical window of exposure with respect to fetal development during weeks 33-40 for the effects of dibromoacetic acid and during weeks 37-40 for the effects of dichloroacetic acid.
Data from 1997-1998 on haloacetic acid (total haloacetic acids, dichloroacetic acid, and trichloroacetic acid), 3-chloro-4-(dichloromethyl)-5-hydroxy-2(5H)-furanone (MX), and mutagenicity were available for a limited number of towns.

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