Graves' disease

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Related to Graves disease: hypothyroidism, Hashimoto's disease

Graves' disease:

see thyroid glandthyroid gland,
endocrine gland, situated in the neck, that secretes hormones necessary for growth and proper metabolism. It consists of two lobes connected by a narrow segment called the isthmus. The lobes lie on either side of the trachea, the isthmus in front of it.
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References in periodicals archive ?
To the best of my knowledge, this is the first description of livedo reticularis in association with Graves disease or hyperthyroidism.
Radiotherapy-induced ophthalmopathy associated with Graves disease most likely results from an autoimmune reaction secondary to a thyroid injury caused by the radiation.
Results for 5 of 6 Graves patient samples provided by Kronus from patients clinically diagnosed with Graves disease were positive (TSH concentrations 370%, 382%, 371%, 262%, and 316%) and 1 was indeterminate (115% of the reference interval TSI value).
Although she denied any symptoms of hyper- or hypothyroidism, laboratory investigations revealed hyperthyroidism and subsequent investigations, including radioactive iodine uptake and scan, suggested Graves disease.
The combination of absent thyroid peroxidase antibodies and high thyroid-stimulating immunoglobulin levels in Graves disease identifies a group at markedly increased risk of ophthalmopathy.
An unusual cause of anterior mediastinal mass: thymic hyperplasia resulting from Graves disease.
The samples included 41 sera from patients with Graves disease (32 untreated hyperthyroid patients and 9 treated patients) and 42 sera from patients with other thyroid diagnoses, as follows: 13 with Hashimoto thyroiditis, 3 with nodules, 13 with multinodular goiter, 3 with cancer, 2 with silent thyroiditis, and 8 with no thyroid disease.
Measurement of Tg in serum is hampered by the presence of circulating interfering factors, especially Tg antibodies (TgAbs), which are frequent in Graves disease and which affect the reliability of Tg assays (8, 13).
It is important to note that the natural course of Graves disease is altered during pregnancy, with an aggravation in the first trimester because of increased thyroid activity, amelioration in the second half of pregnancy because of immune suppression, and aggravation in the postpartum period as the immune system rebounds (29).
125]I-labeled TSH and PEG precipitation (5) in 56 sera from patients suspected of having Graves disease (Fig.
Examples are hypothyroidism with mild, diffuse thyroid enlargement; hyperthyroid Graves disease without ophthalmopathy; or a solitary thyroid nodule.
Autoantibodies to the thyrotropin (TSH) receptor (TSHR) are the hallmark of the autoimmune response to the thyroid gland and are responsible for thyrotoxicosis in Graves disease (1).