mitochondrion

(redirected from Mitochondrial membrane)
Also found in: Dictionary, Thesaurus, Medical.

mitochondrion

a small spherical or rodlike body, bounded by a double membrane, in the cytoplasm of most cells: contains enzymes responsible for energy production
References in periodicals archive ?
Mitochondrial membrane potentials were measured by laser scanning microscopy and comparable results were obtained (Fig.
The use of selected immune-modulating probiotics along with LRT provides the cytokine milieu the opportunity to be beneficially altered through the management of mitochondrial membrane repair, DAMP reduction, and PRR-induced tolerance via changes in bacterial ratios in the gut towards ones that favor symbiosis.
Meanwhile, morphologic changes in the nucleus and cytoskeleton of the treated cells were observed under LCSM with DAPI and tubulin Alexa Fluor 488 staining; whereas, the mitochondrial membrane potential was detected with Mito Tracker Red CMXRos staining.
The collapse of an electrochemical gradient across the mitochondrial membrane during apoptosis was measured using a JC-1 Mitochondrial Membrane, Potential Assay Kit, Cayman Chemical.
By applying established and recently introduced indicators, this integrated approach allows real-time measurement of mitochondrial membrane potential, hydrogen peroxide ([H.
0061551) on a new drug target for type 2 diabetes located in the inner mitochondrial membrane, which includes recently identified proteins that control mitochondrial pyruvate import.
Beta-amyloid treatment also led to the decreased mitochondrial membrane potential, the cleavage of poly(ADPribose) polymerase, an increase in the Bax/Bcl-X(L) ratio, and activation of c-Jun N-terminal kinase.
METHODS: Cell viability, reactive oxygen species (ROS), mitochondrial membrane potential (MMP), cytochrome c (cyt c), phosphorylated extracellular signal-regulated protein kinase (p-ERK), caspases, morphologic change, and functional changes in HEI-OCI cells, rat cochlear explants, and mouse cochlea after [Cd.
However, this protective response was ultimately futile, and the sustained induction of ROS rapidly led to mitochondrial membrane depolarization, release of cytochrome c and activation of caspase-3.
Pretreatment with paeoniflorin prior to MG exposure reduced MG-induced mitochondrial dysfunction by preventing mitochondrial membrane potential dissipation and adenosine triphosphate loss.

Full browser ?