TNF

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TNF

(organic chemistry)
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Our data showed that the concentrations of proinflammatory cytokine TNF-a and CXC motif CXCL8, but not others, were not only significantly elevated in BALF of asthmatics compared with controls, but also are inversely associated with lung function and positively correlated with local infiltration of neutrophils and eosinophils, suggesting the importance of these two molecules in the pathogenesis of asthma.
We had previously shown that TNF-a mRNA-positive cells were significantly elevated in the BALF of asthmatics compared with controls,[sup][14] which was a first report regarding TNF-a in asthma.
In this study, we aimed to assess the significance of two urinary biomarkers; caspase 3 and TNF-a, in patients with UPJO within six months of follow-up.
Also, urinary levels of TNF-a and caspase 3 enzyme were checked in fresh first morning voided urine samples by the use of a commercial enzyme-linked immune absorbent assay kit (RayBiotech, Norcross, GA) and human cysteinyl aspartated specific proteinases 3 (CASPASE-3) ELISA kit (Hangzhou Estabiopharm CO), respectively.
In present study we compared the time and dose dependant expression of TNF-a in head kidney and spleen of Cirrihinusmrigala on stimulation with bacterial and viral synthetic antigens.
The prospective cohort study was designed and done to investigate the serum levels of leptin, ghrelin and TNF-a in patients with cyanotic and acyanotic CHD.
In summary, considering the cited articles [sup][1],[4] and our own study together, we conclude that ethnicity, age, and BMI appear to be important variables influencing the levels of TNF-a and its soluble receptors.
The TNF-a group showed significantly increased apoptosis rates at 12 h and 24 h; meanwhile, a marked reduction of TNF-a induced apoptosis was found in the exenatide group.
HCAECs were treated with 1–10 [micro]mol/L Sal B for 24 h, and then stimulated with 100 ng/ml TNF-a for 10 min (for JNK and ERK1/2), 30 min (for p-I?