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myocardium

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myocardium

the muscular tissue of the heart
Collins Discovery Encyclopedia, 1st edition © HarperCollins Publishers 2005

myocardium

[‚mī·ə′kärd·ē·əm]
(histology)
The muscular tissue of the heart wall.
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
The following article is from The Great Soviet Encyclopedia (1979). It might be outdated or ideologically biased.

Myocardium

 

the muscular layer that constitutes most of the heart. It consists of striated muscle tissue made up of a dense aggregation of muscle cells. A functional characteristic of the myocardium is rhythmic, automatic contraction alternating with relaxation, which continues without interruption throughout the life of the organism.

The Great Soviet Encyclopedia, 3rd Edition (1970-1979). © 2010 The Gale Group, Inc. All rights reserved.
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References in periodicals archive
Increased myocardial gene expression of tumor necrosis factor-alpha and nitric oxide synthase-2: a potential mechanism for depressed myocardial function in hibernating myocardium in humans.
A self-perpetuating vicious cycle of tissue damage in human hibernating myocardium. Mol Cell Biochem 2000; 213:17-28.
Functional cine imaging comparing wall motion at rest and under stress with dobutamine unmasks hibernating myocardium at low doses.
Nevertheless, since areas of hibernating myocardium exist side-by-side with normal heart muscle, the disparity in electrical activity can result in rhythmic chaos, making the heart vulnerable to fatal arrhythmias.
Myocardial cell death and apoptosis in hibernating myocardium. J Am Coll Cardiol 1997;in press.
The precise pathogenesis of stunned and hibernating myocardium remains somewhat controversial and is beyond the scope of this article.
Therefore, a combination of techniques to evaluate several of these parameters is generally used clinically in considering if repetitively stunned or hibernating myocardium exists as a cause of reversible left ventricular dysfunction, warranting coronary revascularization.
The [beta]-blocker also might shift the energy substrate of the myocardium away from free fatty acids and toward glucose; that would further improve efficiency, especially in hibernating myocardium, they said.
Bonow suggested that these patients may have a large amount of hibernating myocardium that is salvaged by revascularization.
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