Gender differences invarious types of idiopathic
ventricular tachycardia. J Cardiovasc Electrophysiol 2002; 13: 633-638.
Catecholaminergic polymorphic
ventricular tachycardia is defined as
ventricular tachycardia that develops in relation with genetic causes and is related with catecholamine release, which is triggered by exertion and emotional stress in the absence of structural cardiac disease (1, 2).
Ventricular tachycardia often develops after injury to the heart, commonly following a heart attack.
Arrhythmias * Non-sustained or sustained
ventricular tachycardia of RV * Non-sustained or sustained outflow configuration left
ventricular tachycardia of left bundle-branch block morphology bundle branch morphology with with the inferior axis (positive superior axis (negative or QRS in leads II, III, and aVF and indeterminate QRS in leads II, negative in lead aVL) or of III, and aVF and positive in unknown axis.
Caption: Figure 1: ECG showing spontaneous subtle change in QRS morphology after induction of
ventricular tachycardia. The first beat ([??]) of tachycardia resembles the clinical VT.
Incidence of and outcomes associated with
ventricular tachycardia or fibrillation in patients undergoing primary percutaneous coronary intervention.
Severely compromised ventricular function, chronic renal insufficiency and
ventricular tachycardia as well as the onset arrhythmia all seem to correlate significantly with the development of storms.
Long-term results of catheter ablation of idiopathic right
ventricular tachycardia. Circulation 1990; 82: 2093-2099, doi: 10.1161/01.CIR.82.6.2093.
A wide QRS tachycardia has a differential diagnosis, but by far the most common cause of a wide complex tachycardia (WCT) is
ventricular tachycardia (VT).
The development of monomorphic
ventricular tachycardia (VT) and/or ventricular premature depolarizations (VPDs) beyond the acute phase following blunt chest trauma is more commonly associated with right or left ventricular structural abnormalities but with little understanding of the underlying mechanisms or recommended therapies.
It explains technical aspects and general concepts of electrophysiologic investigation, sinus node function, atrioventricular conduction, intraventricular conduction disturbances, miscellaneous phenomena related to atrioventricular conduction, ectopic rhythms and premature depolarizations, supraventricular tachycardias, atrial flutter and fibrillation, preexcitation syndromes, recurrent
ventricular tachycardia, the evaluation of antiarrhythmic agents, and catheter and surgical ablation in the therapy of arrhythmias.
Ventricular arrhythmias were present in 32% of them of which 24% had
Ventricular Tachycardia (VT) and 28% had Ventricular Premature Beats (VPB).