ALT 203 U/L (6/50 U/L), AST 180 U-L (35-140 U-L), C-reactive protein (CRP) 3,4 mg/dl (<2 mg/dl); procalcitonine 0.3 ng/ml (<0.5ng/ml) Blood culture Negative Urine culture Negative Cerebrospinal fluid Negative culture Chest X-ray Bilateral pulmonary infiltrates and significant
cardiomegaly Echocardiography Diffuse pericardial effusion affecting predominantly right chambers.
Follow-up chest X-ray showed regression of
cardiomegaly and pulmonary vascular markings.
On chest X-ray, 58.6% VSD cases had
cardiomegaly and plethora.
After 5 weeks of medication, a chest radiograph showed reduced pleural effusion with unchanged
cardiomegaly [Figure 1]c.
[1,2] In the infant under a year, who is a floppy baby in cardiac failure, with
cardiomegaly, the disease should be suspected.
Still further, Waters suggests that both
cardiomegaly and HTN increase the risk for acute PMI since 42% of hearts weighing more than 500 gm, and >1/3 of hypertensive patients had an acutely infarcted papillary muscle.
Chest radiography indicated mild
cardiomegaly. Initially she was treated conservatively with diuretics, digitalis, and penicillin prophylaxis for endocarditis and later scheduled for balloon mitral valvuloplasty.
A 2/6 systolic murmur along the left upper sternal border was heard at physical examination;
cardiomegaly and an increase of pulmonary vascularity were observed with the help of telecardiography.
Examination revealed
cardiomegaly with the apex in the sixth intercostal space in the anterior axillary line.
Chest x-ray showed
cardiomegaly. A non-contrast computed tomography scan of the brain did not show any sign of stroke (haemorrhage).
Mild
cardiomegaly was noted, and there was no evidence of hepatic, splenic, or adrenal involvement.