Cranial neural crest-derived mesenchymal proliferation is regulated by Msx1mediated p19 (INK4d) expression during
odontogenesis. Dev Biol 2003; 261(1): 183-96.
La etiologia de las raices supernumerarias no esta bien esclarecida, se cree que puede deberse a factores externos durante la
odontogenesis o a la penetracion de un gen atavico que se desconoce su origen exacto (6).
Other alterations related to
odontogenesis (amelogenesis imperfecta and dentinogenesis imperfecta) were also considered exclusion criteria.
Expression of FAM20C in the osteogenesis and
odontogenesis of mouse.
Like normal
odontogenesis, the odontogenic tumors represent inductive interactions between odontogenic ectomesenchyme and epithelium (1, 2).
[12-13] In dysmorphic roots, it occurs due to external factors during
odontogenesis or attribution to atavic gene or polygenic system.
Odontogenesis occurs because of a multi-stage complicated interaction between the oral epithelium and the mesenchymal tissue under the epithelium.
The mesenchymal bone marrow stem cells (BMSC) grown together with embryonal epithelial cells of the oral cavity transform into cells similar to odontoblasts, containing the marker protein of
odontogenesis. After grafting bone marrow cells under the renal capsule of adult rats, the development of structures similar to teeth surrounded by soft and bone tissue was observed [46].
They are presumed to arise from the remnants of the dental lamina which persist in the oral tissues after the completion of
odontogenesis, or from the basal cell layer of the oral mucosal epithelium.