acyl-coenzyme A


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acyl-coenzyme A

[′a·səl kō′en‚zim ′ā]
(biochemistry)
References in periodicals archive ?
Long-chain acyl-coenzyme A synthetases (ACSLs) belong to the ACS family and include 5 isoforms, designated ACSL1, ACSL3, ACSL4, ACSL5 and ACSL6, listed in order of their discovery(Kuwata et al.
The expression levels of 10 genes: retinoid X receptor alpha (RXRA), peroxisome proliferator-activated receptor gamma (PPARG), phospholipid transfer protein (PLTP), stearoylCoA desaturase (SCD), nuclear receptor subfamily 1 group H member 3 (NR1H3), fatty acid binding protein 3 (FABP3), carnitine palmitoyltransferase II (CPT2), acyl-Coenzyme A dehydrogenase long chain (ACADL), acyl-Coenzyme A oxidase 2 branched chain (ACOX2), and fatty acid binding protein 4 (FABP4), showed significant differences in gene expression between the low- and high-marbled groups (p < 0.
Purification and properties of very-long chain acyl-coenzyme A dehydrogenase.
Mitochondrial very-long-chain acyl-coenzyme A dehydrogenase deficiency: clinical characteristics and diagnostic considerations in 30 patients.
The cherry diet was also associated with significantly enhanced hepatic PPAR-alpha mRNA, enhanced hepatic PPAR-alpha target acyl-coenzyme A oxidase mRNA and activity, and increased plasma antioxidant capacity.
Medium-chain acyl-coenzyme A dehydrogenase (MCAD) deficiency is a condition which prevents the body from converting certain fats to energy.
The disorders tested in newborn screening include very long-chain acyl-coenzyme A dehydrogenase (VLCAD) deficiency, long-chain 3-hyroxy acyl-coenzyme A dehydrogense deficiency (LCHAD), medium-chain acyl-coenzyme A dehydrogenase (MCAD) deficiency, and short-chain acyl-coenzyme A dehydrogenase (SCALD) deficiency.
Mitochondrial very long-chain acyl-coenzyme A dehydrogenase deficiency: clinical characteristics and diagnostic considerations in 30 patients.
Medium-chain acyl-coenzyme A dehydrogenase deficiency: Clinical course in 120 affected children.
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