angioedema


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Related to angioedema: Hereditary angioedema

angioedema

[‚an·jē·ō·i′dē·mə]
(medicine)
The development of giant hives beneath the surface of the skin, especially around the eyes and lips; usually due to an allergic response.
References in periodicals archive ?
Emergency treatment of acute attacks in hereditary angioedema due to C1 inhibitor deficiency: What is the evidence?
Their multimodal evaluation included the Angioedema Quality of Life questionnaire--a patient-reported, 17-item instrument that is the first validated tool for evaluation of angioedema-specific quality of life--as well as the Dermatology Life Quality Index and the weekly Angioedema Activity Score.
Dr Bruno Giannetti, MD PhD, chief operations officer of Pharming, said, 'hereditary angioedema patients in the US are currently facing a shortage of plasma-derived C1 inhibitor used to prevent attacks.
Angioedema is a multi-etiological condition that can be induced by genetic, infectious, and physical factors, autoimmune disorders, foods, vaccines, insect bites, stings, radiocontrast media, latex, and drugs.
Along with strategically examining the key micro markets, the paper also focuses on industry-specific drivers, restraints, possibilities and challenges in the Angioedema market.
The report provides a snapshot of the global therapeutic landscape of Hereditary Angioedema (HAE) (C1 Esterase Inhibitor [C1-INH] Deficiency)
Acquired angioedema (AAE) differs from HAE by the absence of a family history of AE and a delayed onset of symptoms, usually in the fourth decade of life or later.
57-fold greater risk of angioedema than did those on a non-DPP-IV inhibitor comparator medication.
57-fold greater risk of angioedema than did those on a non-DPP-4 inhibitor comparator medication; however, the absolute risk was very low: 14 confirmed cases in 2,754 subjects.
Angioedema must be suspected in any patient with typical clinical presentations and history of any new drug exposure of similar previous attacks.
ACE-inhibitor-related angioedema is not a true allergy but is considered an adverse class effect secondary to the mechanism of action.
Marc Riedl, Associate Professor and Clinical Director of the US HAEA Angioedema Center at the University of California, San Diego.