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Related to cervicogenic headache: Occipital neuralgia


pain in the head, caused by dilation of cerebral arteries, muscle contraction, insufficient oxygen in the cerebral blood, reaction to drugs, etc.



one of the most frequent symptoms of various diseases.

Headache arises chiefly in response to irritation of the nerve endings (receptors) of the meninges, venous sinuses, and tunicary and cerebral vessels. Leading factors in the development of a headache include inflammatory affection of the meninges (resulting in irritation of the meningovascular receptors by toxins of the pathogenic agent), compression of the receptors by edema in brain tumors and other accumulative processes (for example, abscess, tuberculoma, and cys-ticercosis), impairment of the circulation of the cerebrospinal fluid accompanied by an elevation in intracranial pressure, vascular diseases (hypertension, hypotension, migraine) that cause a change in extracranial and intracranial vascular tone, chronic cardiovascular insufficiency (for example, congestion of the cerebral veins), and infectious diseases and intoxication accompanied by toxic irritation of the nerve endings. Headache also arises through affection of the cervical nerve roots of the spinal cord and affection of the perivascular neuroplexuses of the major blood vessels of the head (for example, in cervical migraine).

Localization of the headache and determination of its character, time of appearance, and other features are important in discovering its causes. Its course and accompanying signs should also be taken into account. Neurological examination and examination of the internal organs, eyes (vision, fundus oculi, intraocular pressure), ears, paranasal sinuses, endocrine glands, and cerebrospinal fluid are also necessary. In a number of cases physicians also make use of cranial roentgenography, electroencephalography, angioenceph-alography or pneumoencephalography, and rheoenceph-alography.

Treatment includes elimination of the cause of the headache and the prescription of preparations (vasodilative or vasoconstrictive, decreasing intracranial pressure [dehydrating], and analgetic) that counteract the cause of the headache.


Platonova, E. P. Golovnye boli. Moscow, 1960.
Botez, M., and R. Şerbánescu. Algille craniene şi craniofaciale: Fiziopatologie, Diagnosti, Tratament. Bucharest, 1965.
Heyck, H. Bóle gtowy. Warsaw, 1960.



A deep form of pain, with a characteristic aching quality, localized in the head.
References in periodicals archive ?
C2/C3 nerve blocks and greater occipital nerve block in cervicogenic headache treatment.
Cervicogenic headache (CGH) is a challenging complaint that is commonly faced by physiotherapists in clinical practice.
To treat the cervicogenic headache, trigger point therapy was performed on the hypertonic suboccipital muscles in sitting.
For the prophylactic treatment of cervicogenic headaches and for acute tension headaches, SMT is superior to placebo.
There were also indications with limited or moderate level evidence that therapeutic exercise was effective for patients with cerebrovascular accident, cerebral palsy, peripheral neuropathy, Parkinson's disease, ankylosing spondylitis, neck pain, whiplash associated disorders, cervicogenic headaches, fractures, shoulder pain, patellofemoral pain, ankle sprains, hypertension, Down syndrome, older adults with health problems, and HIV.
Cervicogenic headaches are unilateral headaches in the second cervical (C2) distribution with no side shift.
Differential diagnoses included cervicogenic headache, migraine headache, and tension-type headache.
Exclusion criteria were a history of vertigo, neuro-musculoskeletal problems or spinal pathologies either acquired or congenital, cervicogenic headache, dizziness during head and neck movements, diabetes, vertebrobasilar insufficiency, visual problems and/or balance disturbances.
This paper has justifiably demonstrated significant short and long-term benefits of a Mulligan self-SNAG in the management of cervicogenic headache.
It has been postulated that noxious stimulation of cervical afferents can result in headache disorders such as migraine and cervicogenic headache.
This article attempts to do this by examining the efficacy of a self-SNAG to C1-C2 in the treatment of cervicogenic headache.