cholinergic

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Related to cholinergic receptors: Nicotinic receptors

cholinergic

[¦kō·lə¦nər·jik]
(physiology)
Liberating, activated by, or resembling the physiologic action of acetylcholine.
References in periodicals archive ?
About Asmacure Asmacure Ltee is a clinical-stage biopharmaceutical company focused on the development of novel, proprietary compounds that target cholinergic receptors for the treatment of inflammation, notably pulmonary airway diseases.
In the study, 21 chronically ill Gulf War veterans and 17 healthy veterans were given small doses of physostigmine, a substance that briefly stimulates cholinergic receptors.
Asenapine binds to several serotonergic, dopaminergic, and a-adrenergic receptors, but has almost no affinity for muscarinic cholinergic receptors, and so is assumed to cause fewer significant anticholinergic side effects, compared with other antipsychotic medications.
The phosphorylated AChE cannot break down ACh and accumulation of acetylcholine in the synaptic cleft causes excess stimulation at cholinergic receptors throughout the central and peripheral nervous system.
Organophosphate pesticides cause decreased brain weight, decreased cholinergic receptors, hyperactivity, motor and coordination problems in laboratory animals.
Muscarinic cholinergic receptors in area postrema and brainstem areas regulating emesis.
Tobacco and alcohol use may be related at least partially because both nicotine and alcohol affect brain nicotinic cholinergic receptors (Collins 1990; Little 2000).
Zarate has pioneered a series of rapidly acting antidepressants by the intravenous route, including the glutamate-active compound ketamine and the blocker of cholinergic receptors scopolamine.
Keywords: Catharanthus roseus roots Catharanthine Ajmalicine Serpentine Acetylcholinesterase Cholinergic Receptors
Neurotoxic effects result when reactive oxon metabolites form covalent bonds with acetylcholinesterase (AChE) and inhibit AChE-catalyzed inactivation of the neurotransmitter acetylcholine, resulting in hyperstimulation of cholinergic receptors and symptoms of acute OP toxicity.