Tex6 and Oh516 had highly significant and negative GCA for ear rot
and aflatoxin concentration, indicating that they should contribute resistance for both traits (Table 2).
Low: accessions with ear rot
values of 1 in both environments (1,1) or i in one environment and 2 in the other (1,2 in any order).
1]), ear rot
(%), erect plants (%), and moisture (%) calculated for each accession.
01) for yield, days to silk, ear height, ears per plant, and ear rot
(Tables 1 and 2).
Inheritance of resistance to aflatoxin production and Aspergillus ear rot
of corn from the cross of inbreds B73 and Oh516.
Corn ear rot
caused by Fusarium moniliforme is a destructive disease for the decreasing yield of corn (Bezuidenhout et al.
48 Source of % Cob % Ear rot
Segr variation Genotypes (G) 7.
1%, mighty play an important role in the control of stem and ear rot
Husk coverage and ear declination in relation to corn ear rots
353-361, the estimates of heritability for fumonisin concentration and Fusarium ear rot
in the NC300 x B104 recombinant inbred population were computed incorrectly.
Fumonisins are produced by the corn ear rot
fungus Fusarium verticillioides, which is one of the most common pathogens of corn worldwide.
Fusarium ear rot
(FER) disease in maize reduces grain quality and yield to an appreciable extent.