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Any disease of the brain.



a collective term that designates a noninflammatory organic disease of the brain. Some encephalopathies are innate, resulting from embryopathy. Others are the result of infection, poisoning, trauma, or vascular disease of the brain. There are no specific manifestations. The most common encephalopathies resemble neuroses (asthenia, irritability, insomnia, headaches) or psychoses (narrowed scope of interests, passivity, emotional instability, vulgarity). Symptoms may include memory loss or mental deterioration.

Alcoholic encephalopathies are alcoholic psychoses. They may be acute, as in Wernicke’s encephalopathy (named for the German neuropathologist C. Wernicke, who described the condition in 1881), or chronic, as in Korsakov’s psychosis (named for S. S. Korsakov) and alcoholic pseudoparalysis. Lead encephalopathy is caused by chronic poisoning by lead salts.

Treatment for encephalopathies depends on the cause of the disease.

References in periodicals archive ?
Hallmarks of HACE are encephalopathic symptoms and signs, including ataxic gait severe lassitude, and progressive decline of mental function and consciousness (irritability, confusion, impaired mentation, drowsiness, stupor, and finally coma).
Elder child was diagnosed after encephalopathic crisis and suffers severe MD.
9%), consistent with a previously unknown diffuse encephalopathic process from drugs, alcohol, an environmental toxin or from insufficiently addressed medical problems.
Serious complications, such as severe and sometimes fatal encephalopathic adverse reactions and coma in patients with onchocerciasis combined with high intensity of Loa loa have been reported (Chippaux et al.
A recent viral infection or vaccination usually induces the characteristic multifocal clinical presentation with encephalopathic symptoms (e.
Calcification of the basal ganglia in encephalopathic HIV/AIDS children has been relatively well documented.
None of the patients was encephalopathic or postoperative; all were pharmacologically paralysed.
Underlying medical conditions were present in 55% of the 38 encephalopathic children, of whom 32% had neurologic or developmental disorders.
The agent chosen was oral succimer (dimercaptosuccinic acid) because the patient was not severely encephalopathic.
Standard parenteral nutrition formulations can be initiated if the patient is not encephalopathic at baseline (Driscoll & Blackburn, 1990).
8] Additionally, acellular vaccines reportedly are associated with less severe life-threatening reactions including shock, collapse, and encephalopathic reactions.