glucose-6-phosphate


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Related to glucose-6-phosphate: glucose-6-phosphate dehydrogenase, Glucose-6-phosphate dehydrogenase deficiency, glucose-6-phosphate dehydrogenase test

glucose-6-phosphate

[′glü‚kōs ¦siks ′fäs‚fāt]
(biochemistry)
C6H13O9P An ester of glucose with phosphate attached to carbon atom 6. Also known as Robisonester.
References in periodicals archive ?
Predictive role of laboratory and clinical treatment response parameters and glucose-6-phosphate dehydrogenase status in the therapy of falciparum malaria.
An improved, simple screening method for detection of glucose-6-phosphate dehydrogenase deficiency.
Gilbert syndrome and glucose-6-phosphate dehydrogenase deficiency: a dose-dependent genetic interaction crucial to neonatal hyperbilirubinemia.
Copper overload may result from intentional or accidental ingestion and hemolysis, such as glucose-6-phosphate dehydrogenase deficiency hemolysis (19), in which cases nonceruloplasmin copper is greatly increased in plasma (14).
Molecular heterogeneity of glucose-6-phosphate dehydrogenase (G6PD) variants in Italy.
by NADPH-dependent nitrate reductase (incubated with glucose-6-phosphate dehydrogenase and NADPH in 14 mmol/L sodium phosphate buffer, pH 7.
Reaction mixture used was a NADPH generator system consisting of: NADPH, glucose-6-phosphate, glucose-6-phosphate dehydrogenase and magnesium chloride.
The activities of hexokinase, pyruvate kinase and glucose-6-phosphate dehydrogenase were significantly decreased whereas the activities of lactate dehydrogenase.
Dabrafenib can cause serious side effects, some of which can be life threatening, including increasing the risk of developing new primary cutaneous malignancies (new skin cancers), tumour promotion in BRAF wild-type melanoma, serious febrile drug reactions (severe fevers), hyperglycaemia (blood sugar problems), uveitis and iritis (severe eye problems), haemolytic anaemia in patients with glucose-6-phosphate dehydrogenase (G6PD) deficiency and embryofoetal toxicity (potential harm to the unborn baby in pregnant women).
Its failure to combat cyanosis should suggest the possibility of glucose-6-phosphate dehydrogenase (G6PD) deficiency or nicotinamide-adenine dinucleotide phosphate, reduced form (NADPH)-methaemoglobin reductase deficiency (3).
Other groups especially susceptible to metHb formation include pregnant women and people deficient in glucose-6-phosphate dehydrogenase or metHb reductase.
In the 1950s, it emerged that an inherited deficiency of glucose-6-phosphate dehydrogenase, a red blood cell enzyme, caused this reaction, known as favism.

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