glycogenolysis


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glycogenolysis

[′glī·kə·jə′näl·ə·səs]
(biochemistry)
The metabolic breakdown of glycogen.
References in periodicals archive ?
Beta-blockers antagonize hepatic glycogenolysis and blunt the sympathetic nervous system responses of associated with hypoglycemia.
27, 28] Direct action of insulin on hepatocytes activates insulin receptor substrate phosphatidylinosital 3-OH [IRS-PI (3)K], which in turn activates Akt that inhibits Forkhead box protein 01 (Fox01), a transcription factor that stimulates gluconeogenesis and glycogenolysis, the two determinants of HGP.
1988) Role of cyclic AMP and inorganic phosphate in the regulation of muscle glycogenolysis during exercise.
Stress-related hormones also increase the blood glucose concentration through mobilized energy reserves, such as tissue glycogen through glycogenolysis (MORMEDE et al.
The subjects performing with water might have depended for glucose on the internal mobilization of muscle glycogen, glycogenolysis, lactate conversion and free fatty acids.
Regulation of hepatic gluconeogenesis and glycogenolysis by catecholamines in rainbow trout during environmental hypoxia.
Release of adrenaline and noadrenaline during the initial stage of stress may also stimulate hepatic glycogenolysis leading to hyperglycemia (Knowles and Warriss, 2000).
3] receptor binding TCDD AhR, thyroid hormone Nicotine Suppresses glycolysis and glycogenolysis, reduces synthesis of phosphorylase and phosphofructokinase, inhibits [Na.
The inhibitory actions include suppressing gluconeogenesis, lipolysis, glycogenolysis, ketogenesis and proteolysis (4,12).
Some metabolic mechanisms include: i) stimulation of glycogenolysis, providing substrate for anaerobic glycolysis and increasing ATP production to meet utilization; ii) regulation of the absolute activities in the pathways of both aerobic and anaerobic metabolism; and iii) regulation of the ratios of glycolytic/oxidative capacities on a tissue-by-tissue basis (Almeida-Val and Hochachka, 1995; Storey, 1996).
Fructose 1-phosphate inhibits glucose formation by inhibiting gluconeogenesis and glycogenolysis, thereby promoting systemic hypoglycemia and ATP depletion.