carcinoma

(redirected from hepatocarcinoma)
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Related to hepatocarcinoma: hepatocellular carcinoma

carcinoma:

see neoplasmneoplasm
or tumor,
tissue composed of cells that grow in an abnormal way. Normal tissue is growth-limited, i.e., cell reproduction is equal to cell death. Feedback controls limit cell division after a certain number of cells have developed, allowing for tissue repair
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.

carcinoma

[‚kärs·ən′ō·mə]
(medicine)
A malignant epithelial tumor.

carcinoma

Pathol
1. any malignant tumour derived from epithelial tissue
2. another name for cancer
References in periodicals archive ?
Furthermore, we have demonstrated a promising cytotoxic potential for Ci and Vc towards human hepatocarcinoma and breast adenocarcinoma cell lines whose mechanisms of action deserve further investigation.
Sex hormone-binding globulin secretion by human hepatocarcinoma cells is increased by both estrogens and androgens.
Infectivity of the monkey HEV strain was examined ex vivo with a human hepatocarcinoma cell line (PLC/PRF/5), and in vivo with 2 HEV-negative cynomolgus monkeys.
Toxicologic studies have reported that chronic exposure to PFCs, including PFOS and perfluorooctanoic acid (PFOA), which is not technically a POP because of its short half-life, can lead to hepatotoxicity, including increased liver weight, fluctuations in liver enzyme levels, and hepatocarcinoma (Son et al.
Lipid hydroperoxides from processed dietary oils enhance growth of hepatocarcinoma cells.
The activity of AEG35156 is currently being explored, in combination, or as a monotherapy, in the following indications: acute myeloid leukemia, non-small cell lung cancer, hepatocarcinoma, and chronic lymphocytic leukemia and indolent B-cell lymphomas.
The MAT preclinical pipeline contains product candidates for acute myeloid leukemia, chronic lymphocytic leukemia, hepatocarcinoma, pancreatic cancer and metastatic melanoma.
Because the genes regulated by hepatoprotectants are relevant to antioxidant or antiapoptotic pathways, we proposed that these compounds exerted their hepatoprotective effects by regulating apoptosis and oxidative stress in hepatocarcinoma cells.
patients Sex M 459 F 241 Age at transplantation, y <52 347 [greater than or equal to] 52 363 Transplant type Liver 171 Kidney 529 Cause of kidney transplantation Glomerulonephritis 188 Genetic nephritis 102 Pyelonephritis or interstitial nephritis 92 Nephroangiosclerosis 82 Other 57 Cause of liver transplantation Alcoholic cirrhosis 67 Hepatitis B or C 60 Autoimmune cirrhosis 9 Other 35 Hepatocarcinoma (yes/no) 64/107 Induction treatment (yes/no) 501/199 Interleukin-2 receptor blockers 292 Rabbit antithymocyte globulins 160 Immunosuppressive therapy at discharge Belatacept 49 Cyclosporine A 182 Tacrolimus 442 Steroids 657 Mycophenolate 593 Azathioprine 2 HEV IgG positive, Characteristic no.
Furthermore, there is some discrepancy in the cellular metabolic ability against chemicals between CHO-K1 and other cell lines such as hepatocarcinoma cells.