Myopericarditis was suspected because of the TnI elevation in the laboratory results, hypokinesis
of the anterior wall, and minimal pericardial effusion identified via echocardiography.
In the mid- and basilar-inferior septum, inferior wall, and posterior-lateral wall, < 50% subendocardial DHE with corresponding mild hypokinesis
and fixed-perfusion reststress defects, corresponds to RCA scar, but viable myocardium, related to the prior MI.
Ventriculography after the operation (B) demonstrated apical ballooning and hypokinesis
with preserved basal contraction.
A transthoracic echocardiogram (TEE) showed hypokinesis
of the basal segments of the anterior, inferior, and lateral walls with preserved function in the apical segments which is opposite of the findings in takotsubo cardiomyopathy.
and functional electric stimulation during standing in persons with spinal cord injury.
An echocardiogram showed ventricular septal hypokinesis
with an ejection fraction of 60%.
A transthoracic echocardiogram revealed left ventricular systolic dysfunction with global hypokinesis
and an ejection fraction of 30% (reference range 55-70%).
Ventriculography showed distorted contraction of the postero-basal segment (1-3) and occasionally anterior hypokinesis
(2, 4), that was related to the rigidity and immobilization of mitral valve complex, proposed to be due to scarring of the mitral valve complex and fibrosis of the papillary muscle.
Transthoracic echocardiography demonstrated severe left ventricular dysfunction, with hypokinesis
of the anteroseptal ventricular wall.
There was akinesis of the inferior wall and significant hypokinesis
of the anterolateral wall.
Stenotic arteries are unable to meet the requirements of increased myocardial oxygen demand to the same extent as a non-stenotic artery, resulting in hypokinesis
of the myocardial segment supplied by the diseased artery with reduced myocardial contractile reserve.
A subsequent bedside transthoracic echocardiogram (TTE) showed severe segmental left ventricular systolic dysfunction with akinesis of the middle and distal segments of the left ventricle and hypokinesis
of the mid and distal inferior walls.