hyperphosphaturia

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Related to hypophosphatemia: hypophosphatasia

hyperphosphaturia

[¦hī·pər‚fäs·fə′tu̇r·ē·ə]
(medicine)
An excess of phosphates in the urine. Also known as phosphaturia.
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In contrast, no statistically significant differences were found for the following variables: gender, BMI, cigarette smoking, HCV or HBV infection, diabetes mellitus, hypophosphatemia, or hyperparathyroidism.
3) Certain electrolyte derangements also may be a cause rather than a sequela of rhabdomyolysis and include hypophosphatemia as well as hyponatremia, a documented cause of rhabdomyolysis in some human patients with psychogenic polydipsia and congestive heart failure.
Biopharmaceutical company Ultragenyx Pharmaceutical (NasdaqGS:RARE) reported on Thursday the completion of adult patient enrollment under the Phase 3 study of KRN23 for the treatment of adults with X-linked hypophosphatemia (XLH), with expected clinical data in 2017.
Seven patients (33%) experienced treatment-related Grade 3-4 adverse events, including: anemia, aseptic meningitis, autoimmune hepatitis, generalized rash, headache, hyperglycemia, hypoglycemia, hypophosphatemia, increased alanine aminotransferase, increased aspartate aminotransferase, increased gamma-glutamyltransferase, muscle spasms, macular rash, rash, and pneumonitis.
18,19) Although there was significant improvement in pain and bone markers with use of denosumab, authors suggested that physicians should carefully consider biochemical and hormonal changes including hypophosphatemia, secondary hyperparathyroidism, and hypercalcemia.
6) Signs of proximal renal tubular dysfunction include proteinuria, glucosuria, hypophosphatemia, hypouricemia, hypokalemia, low serum bicarbonate and/or elevated serum creatinine.
Additional risk factors include chronic alcoholism, statin intake, hypokalemia, hypernatremia, and hypophosphatemia [3].
All laboratory values are within normal limits except for mild hypophosphatemia (3.
Hungry bone syndrome (HBS) refers to a severe, rapid and prolonged hypocalcemia associated with hypophosphatemia and hypomagnesaemia that are the consequences of suppressed parathyroid hormone levels at patients with prior high bone turnover [1-6, 9, 11, 12].
Acute kidney damage secondary to rhabdomyolysis caused by acute hypophosphatemia.
Some of these clinical portrayals testified that the myopathy could be autonomous of metabolic variations from the norm, for example, hypocalcemia [13] and hypophosphatemia [15].