melanogenesis


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melanogenesis

[‚mel·ə·nō′jen·ə·səs]
(biochemistry)
The formation of melanin.
References in periodicals archive ?
In lesional skin, this therapy decreases Langerhans cell activation and increases apoptosis, melanocyte proliferation, and melanogenesis.
High performance liquid chromatography analysis of eu- and pheomelanin in melanogenesis control.
Non-genetic factors such as aging, ultraviolet (UV) light, chronic inflammation, and hormonal changes are also involved in melanogenesis (Costin and Hearing, 2007).
Studies suggested that it regulates the melanogenesis through binding to a highly conserved M-box (GTCATGTGCT) and E-box (TCATGTG) motif upstream of the tyrosinase promoter, which strongly stimulates the tyrosinase promoter activities and promotes tyrosinase expression (Yasumoto et al.
Tyrosine-induced melanogenesis shows differences in morphologic and melanogenic preferences of melanosomes from light and dark skin types.
Melanogenesis is a biochemical process that stimulates the production of eumelanin, the dark pigment of the skin which is known to protect the body from skin damage as a result of exposure to both UV-A and UV-B radiation.
Ceramides inhibit Melanogenesis, which results in age spots and skin discoloration.
ABCB5 is involved in melanin transport during melanogenesis and is highly expressed in melanoma (Chen et al.
2012b) suggested phosvitin as melanogenesis inhibitor.
The effect of alpha-methyldopa and ultraviolet irradiation on melanogenesis.
Inhibitory effect of magnesium L-ascorbyl-2-phosphate (VC-PMG) on melanogenesis in vitro and in vivo.