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myocardial infarction |
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myocardial infarction: see under infarction myocardial infarction or heart attack is usually caused by a blockage in one of the coronary arteries that supply blood to the heart muscle. The blockage typically occurs when a blood clot (see thrombosis ) lodges in an area already narrowed by arteriosclerosis ; other ..... Click the link for more information. . heart attackor myocardial infarctionDeath of a section of heart muscle when its blood supply is cut off, usually by a blood clot in a coronary artery narrowed by atherosclerosis. Hypertension, diabetes mellitus, high cholesterol, cigarette smoking, and coronary heart disease increase the risk. Symptoms include severe chest pain, often radiating to the left arm, and shortness of breath. Up to 20% of victims die before reaching the hospital. Diagnosis is done by electrocardiography and by analysis for enzymes in the blood. Treatment aims to limit the area of tissue death (infarct) and prevent and treat complications. Thrombolytic (clot-dissolving) drugs may be administered. Beta-blockers alleviate pain and slow the heart rate. Angioplasty or coronary bypass restores blood flow to heart muscle. Follow-up may include drugs, exercise programs, and counseling on diet and lifestyle changes. |
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| AMR-001 is an autologous bone marrow-derived cell product being tested to determine if its infusion into the coronary (heart) artery after myocardial infarct can restore heart muscle function in tissue damaged by the heart attack. JETSTENT, AngioJet Rheolytic Thrombectomy Before Direct Infarct Artery Stenting in Patients Undergoing Primary PCI for Acute Myocardial Infarction, will measure final myocardial infarct size in patients with angiographically evident thrombus who receive interventional treatment for acute myocardial infarction, comparing patients treated with Angiojet(R) thrombectomy prior to direct stenting to patients treated with direct stenting alone. The approval came on the heels of years of clinical trials showing that the standardized extract significantly reduced myocardial infarct size (the portion of the heart muscle that is damaged by insufficient blood supply) and lessened myocardial injury, lowered the oxygen consumption of the heart muscle in tissue and animal studies, and reduced platelet aggregation (blood cell stickiness) and thrombosis (blood clot) formation. |
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