natriuresis


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Related to natriuresis: aldosterone, Atrial natriuretic peptide, Natriuretic peptide, pressure natriuresis

natriuresis

[¦na·trē·yu̇′rē·səs]
(physiology)
Excretion of sodium in the urine.
References in periodicals archive ?
Several studies have demonstrated that the slope of the normal chronic salt-loading pressure natriuresis curve is very steep.
How this peptide causes renal sodium excretion has yet to be to be elucidated, but it was determined that infusing digoxin-specific antibodies directly into the ventricles of the rat brain, blocks the central nervous system response to natriuresis (Wijdicks, Vermulean, van Brummelen, den Boer, & van Gijn, 1987).
Pressure-diuresis (P/D) and pressure natriuresis (P/N) responses: In the absence of any significant changes in MAP before and following ARD, significantly (P<0.
Thiazide diuretics lower BP through natriuresis (ie, sodium excretion), resulting in volume depletion and reduction of peripheral vascular resistance over time.
How could a naturally occurring peptide that promotes vasodilation, facilitates natriuresis, and reduces neurohormonal activation be unsafe?
These results indicate that chronic salt-loading pressure natriuresis curve plays a central role in long term control of arterial blood pressure and the development of DOCA-salt hypertension.
ANP is involved in natriuresis, diuresis, vasodilation and cardiovascular homeostasis.
Plasma ADM concentrations reflect activation of defense mechanisms against vascular damage, besides vasodilation and natriuresis, such as antiinflammation and antioxidation, and exhibit proliferative effects on vascular endothelial cells and antiproliferative effects on vascular smooth muscle cells, supporting the role of ADM as an antiproliferative factor inhibiting the development of atherosclerosis (7).
The major finding of the present study is the demonstration that ARD-induced diuresis and natriuresis can possibly be accompanied by a prominent enhancement in whole kidney GFR even when basal renal haemodynamic responses remain unaltered.
BNP results in natriuresis, diuresis, vascular smooth muscle relaxation, and inhibition of the renin-angiotensin system (RAS).
The roles of natriuretic peptides in promoting vasodilation and natriuresis are well known (1, 2), and assays for both the active form of B-type natriuretic peptide (BNP) [6] and the inactive amino-terminal fragment of the peptide (NT-proBNP) have gained widespread acceptance as tools for the diagnosis of heart failure in the acute-care setting (3, 4).