microangiopathy

(redirected from thrombotic microangiopathy)
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microangiopathy

[‚mī·krō‚an·jē′äp·ə·thē]
(medicine)
The development of lesions in small blood vessels throughout the body.
References in periodicals archive ?
Not only have genetic or acquired derangements in ADAMTS13-mediated cleavage of von Willebrand Factor been described as the underpinnings of thrombotic microangiopathy (as in TTP), but also complement-, toxic-, drug-, coagulation-, and metabolicmediated pathophysiologies.
Secondary thrombotic microangiopathy can be induced by a large number of disorders such as malignancy, infection, disseminated intravascular coagulopathy, autoimmunity, chemotherapy drugs, and rarely sickle cell crisis.
The clinical findings in hemolytic uremic syndrome occur as a result of thrombotic microangiopathy (TMA).
and the European Union as the first and only treatment for patients with atypical hemolytic uremic syndrome (aHUS), a debilitating, ultra-rare and life-threatening genetic disorder characterized by complement-mediated thrombotic microangiopathy, or TMA (blood clots in small vessels).
The clinical pictures were consistent with thrombotic microangiopathy and are the first reported cases of tiger snake envenomation associated thrombotic microangiopathy.
Thrombotic microangiopathy (TMA) is an important histological finding in catastrophic APS (CAPS) and in APS patients with nephropathy.
Thrombotic microangiopathy in malignant hypertension and hemolytic uremic syndrome (HUS)/ thrombotic thrombocytopenic purpura (TTP): can we differentiate one from the other?
Endothelial lesions: thrombotic microangiopathy (TMA) also occurs at elevated rates among patients with HIV (13)
Manifestations include renal thrombotic microangiopathy, respiratory distress syndrome (ARDS) and disseminated intravascular coagulation.
TTP is one of the conditions in the broad spectrum of thrombotic microangiopathy (TMA) that is characterized by the formation of thrombi in the microvasculature of various organs and has remained a syndrome with multiple unknowns until recently.
A genetic form of thrombotic microangiopathy, known as Factor H associated HUS, is characterized by deficient quantities of factor H and uncontrolled complement activation.