Vaginitis


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Related to Vaginitis: Yeast infection, trichomoniasis, clotrimazole, cervicitis

vaginitis

[‚vaj·ə′nīd·əs]
(medicine)
Inflammation of the vagina.
Inflammation of a tendon sheath.

Vaginitis

 

(colpitis), an inflammation of the mucous membrane of the vagina. Vaginitis is often combined with inflammation of the external genitalia; that is, it may occur as vulvovaginitis.

Vaginitis is caused by the penetration into the vagina of various microbes or parasites (gonococci, trichomonads, and pinworms) from contaminated underclothing or dirty hands or after failure of one partner to observe the rules of sexual hygiene, and so forth. It may also occur after prolonged mechanical irritation of the mucous membrane (for example, prolonged wearing of a girdle for prolapse of the uterus). Vaginitis may develop in girls age three to ten when the vagina becomes infected by the blood flow (in diphtheria, scarlet fever, measles, and other diseases).

The symptoms of the disease include swelling and reddening of the mucous membrane of the vagina, purulent leukorrhea (sometimes with an admixture of blood), a sensation of heaviness in the lower abdomen, a burning sensation, and pruritus of the external genitalia. Among the symptoms of vulvovaginitis are a reddening of the external genitalia that often spreads to the thighs and buttocks, purulent discharges, and pruritus. Senile vaginitis may arise after age-related changes (shriveling and dryness of the mucous membrane of the vagina). Treatment involves removal of the causes of vaginitis. In cases of trichomonad vaginitis both spouses are usually treated at the same time.

Vaginitis in animals results from injury to the vagina during labor and mating as well as from the penetration of pathogenic microorganisms into the vaginal mucous membranes. The course of vaginitis may be acute or chronic. The vaginal mucous membrane in sick animals is edematous and hemorrhagic. In suppurative vaginitis, the body temperature is raised, urination becomes painful, the animal’s general condition deteriorates, and, in cows, the milk yield declines. Putrescent vaginitis often ends in death. Vaginitis, especially if chronic, may impair the animal’s reproductive capacity. Treatment involves washing the vagina with disinfectants and irrigation with antibiotics. Vaginitis may be prevented by the observance of hygienic regulations during parturition, mating, and artificial insemination of animals.

REFERENCES

Studentsov, A. P. Veterinarnoe akusherstvo i ginekologiia, 3rd. ed. Moscow, 1961.
Gubarevich, la. G. “Vaginit.” In Veterinarnaia entsiklopediia, vol. 1. Moscow, 1968.
References in periodicals archive ?
Among the girls with vulvar erythema, the prevalence of vestibular bands was almost 86%, while among those without vaginitis the prevalence of bands was just over 18%, a statistically significant difference.
In a clinical study published in the May issue of the journal Menopause (ii), VAGIFEM(TM) (estradiol vaginal tablet) was reported to be as effective in treating postmenopausal atrophic vaginitis as the most commonly used estrogen-containing vaginal cream, Premarin(R) Vaginal Cream (PVC).
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Paul Lawrence, Litmus' Chairman and Chief Technology Officer stated "Traditional methods used to detect infectious vaginitis, and specifically bacterial vaginosis are subjective and misdiagnosis is common.
We have a crisis in the diagnosis of candidal vaginitis.
This new finding that patients with relapsing candidal vaginitis have a local allergic response as reflected by the presence of immunoglobulin in their vaginal fluid raises the intriguing possibility that they might benefit from desensitization therapy Dr.
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Atrophic vaginitis has been recognized as a diagnostic and clinical entity since the inception of cervical cancer screening, which serendipitously emerged through the investigation of hormonal influences on the cervix and vaginal vault.
Atrophic vaginitis is irritation of the delicate tissues internally.
A retrospective review of patients referred to the Vaginitis Clinic between 2000 and 2010 revealed 25 patients with clinically refractory fluconazole-resistant vulvovaginitis with confirmed in vitro resistance, with an MIC at least 2 meg/mL (median 8 meg/mL).
Following coverage of the basic physiology of the intestinal microecology, international specialists review the literature on the growing clinical use of probiotics and make recommendations for use in diarrheal diseases, inflammatory bowel disease, irritable bowel disease, treatment/prevention of surgical infections, as an adjunct/alternative to antibiotics, vaginitis, and more controversially, allergic disorders.
2) The role of Group B streptococci (GBS) as an etiologic agent of vaginitis in adults has not been established; it is considered a normal inhabitant of the adult vaginal flora and is not associated with an inflammatory response.