Relationships between tissue levels of 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD), mRNAs, and toxicity in the developing male Wistar(Han) rat.
The importance of pharmacokinetics in determining the relative potency of 2,3,7,8-tetrachlorodibenzo-p-dioxin
Persistent suppression of delayed-type hypersensitivity in adult F344 rats after perinatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin
In utero exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin
causes accelerated terminal differentiation in fetal mouse skin.
induced alterations in protein phosphorylation in guinea pig adipose tissue.
(TCDD) alters pancreatic membrane tyrosine phosphorylation following acute treatment.
KEY WORDS: acetylcholinesterase (AChE), aryl hydrocarbon receptor (AhR), dioxin-responsive element (DRE), neuron, 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD), transcriptional regulation.
The relevance of fat content in toxicity of lipophilic chemicals to terrestrial animals with special reference to dieldrin and 2,3,7,8-tetrachlorodibenzo-p-dioxin
Mortality rates among trichlorophenol workers with exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin
Glucocorticoid potentiation of cytochrome P4501A1 induction by 2,3,7,8-tetrachlorodibenzo-p-dioxin
in porcine and human endothelial cells in culture.
Polychlorinated dibenzodioxins (PCDDs), dibenzofurans (PCDFs), and biphenyls (PCBs) constitute a group of widespread and persistent organic pollutants with 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD, dioxin) being the most toxic member of this group (Birnbaum 1994b, 1995a, 1995b).
POPs include dioxins and furans--with 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD) being the most potent of these and polychlorinated biphenyls (PCBs).