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Figure 1 shows unadjusted Kaplan-Meier cumulative incidence estimates for all-cause mortality according to APOE genotype and allele.
In conclusion, results of our study suggest that polymorphism of ApoE genotype might be a marker that is associated with the onset of disease in patients with non-lesional TLE.
studied the relationship between T2DN and ApoE genotype and found that the ApoE [epsilon]4 allele frequency was significantly higher in stable renal function group (17.1%) than in the microalbuminuria group (8.9%) (P = 0.03); however, ApoE [epsilon]3 allele frequency was significantly higher in proteinuria group than in stable renal function group[29].
It is worth mentioning that 12.0% of our control population had APOE genotypes carrying [epsilon]4 allele ([epsilon]3[epsilon]4, [epsilon]2[epsilon]4) and 50.0% of LOAD patients had APOE genotypes not carrying [epsilon]4 allele ([epsilon]3[epsilon]3, [epsilon]2[epsilon]3).
We aimed to assess APOE genotypes association with myocardial infarction in Pakistani cohort.
Rimbach, "Impact of apoE genotype on oxidative stress, inflammation and disease risk," Molecular Nutrition and Food Research, vol.
We further tested whether there were statistical interactions between rs10550296 genotype and age (categorised age [less than or equal to] 72 years and > 72 years), gender, and ApoE genotype, but no significant interaction was found (data not shown).
"The findings support our hypothesis that APOE genotype changes amyloid structure.
Association of APOE genotype with carotid atherosclerosis in men and women: the Framingham Heart Study.