angiotensin

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angiotensin

[‚an·jē·ə′ten·sən]
(biochemistry)
A decapeptide hormone that influences blood vessel constriction and aldosterone secretion by the adrenal cortex. Also known as hypertensin.
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
The results showed that the level of vitamin D was related to many pathological aspects of respiratory distress at the molecular level because it influenced the amount of specific proteins in ATII cells [28].
The studies included animal and human patients and assessed vitamin D status and its many associations with ATII cells.
According to our previous study, ATII cells were transfected at 70% confluence with shRNA-TTF-1 (336312, Qiagen, Valencia, CA) or shRNA-scrambled (sc-108060, Santa Cruz Biotechnology, CA, USA) [26].
ShRNA-TTF-1-transfected and nontransfected ATII cells were pretreated with liraglutide (100nM) for 4h and then stimulated with LPS (100ng/mL) for 4h [21, 26, 27].
The results of this study disclose behaviours of miR-19a and miR-20a potentially involved in the posttranscriptional regulation of TF expression in the context of the complex interplay among inflammation, coagulation, and ATII, in which TF plays a pivotal role.
Abbreviations Ag: Antigen ATII: Angiotensin II AT1R: Angiotensin II type 1 receptor AU: Arbitrary units CG: Control glucose ELISA: Enzyme-linked immunosorbent assay HG: High glucose miR: MicroRNA MTT: Dimethyl thiazolyl diphenyl tetrazolium OLM: Olmesartan PBMC: Peripheral blood mononuclear cell PCA: Procoagulant activity qRT-PCR: Quantitative real time polymerase chain reaction RAS: Renin angiotensin system RNA: Deoxyribonucleic acid ROS: Reactive oxygen species RPMI: Roswell Park Memorial Institute SD: Standard deviation TF: Tissue factor.
Villagers typically call out "Hi, atii!" in greeting a namesake as they go about their daily affairs in the village.
An Elder answered my question about whether he remembered being nuniaq-ed by speaking of the association with his atii's family:
Furthermore, it has also been demonstrated that treatment with LXA4 could upregulate the CFTR protein expression in vivo and in primary ATII cells.
RvD1 could increase Na + currents in primary ATII cells, and enhance the subcellular distribution of ENaC and Na, K-ATPase.
The differences between treatments versus controls were significant for MX, HHCB, and ATII (p < 0.05, paired t-test), and there were clear trends of increased rhodamine levels also for MK, ADBI, and AHTN, indicating an inhibitor-related effect after 24 hr (Figure 4).
Additionally, we also investigated its effects on the CFTR protein expression in the rat lungs and primary ATII cells.