Most studies suggest that diuretics cannot improve the prognosis of AKI patients, and may even worsen kidney damage, so diuretics are not recommended.12,13 However, for AKI patients with excessive volume overload, furosemide can be used to alleviate excessive volume overload, improve pulmonary edema caused by AKI, reduce the frequency of RRT application and treatment costs, shorten hospitalization time, alleviate injury to kidney function, and help patients go through dangers.14,15 Thus, furosemide is widely used in patients with early AKI and acute pulmonary edema
who cannot be performed with RRT for various reasons.
In our case, the patient presented to the emergency department with unexplained acute pulmonary edema
. His B-type natriuretic peptide level, renal function test results, and echocardiography results were all within the normal range, thus excluding the possibilities of intravascular fluid overload, renal failure, and impairment of cardiac contractility.
In that same study, the symptoms of maternal edema and weight gain were the most common (89.3%), followed by increased blood pressure (60.7%), mild anemia and hemodilution (46.4%), proteinuria (42.9%), oliguria (16.1%), and acute pulmonary edema
(21.4%), all of which were present in the patient's medical chart.
We report here an unusual presentation of a case of severe scorpion envenoming complicated by acute pulmonary edema
and cardiogenic shock in an adult patient, with the presence of very rapid sustained ventricular tachycardia during the early phase.
Acute pulmonary edema
is uncommon, but may be due to a reaction directly in the airways and lungs, or it may reflect cardiac decompensation and/or myocardial infarction.
Acute pulmonary edema
in EV71 infections was rarely reported before the 1998 outbreak in Taiwan (12).
The pathogenesis of acute pulmonary edema
associated with hypertension.
Flash pulmonary edema and acute pulmonary edema
are terms used to define the sudden development of respiratory distress related to the rapid accumulation of fluid within the lung interstitium secondary to elevated cardiac filling pressures (Little, & Braunwald, 1997).
Hospitalization with acute pulmonary edema
was an important marker for increased mortality risk in patients with ischemic HF, but not in those with hypertensive HF.
The incidence of acute pulmonary edema
and arrhythmias was significantly lower in the coenzyme Q10 group.
A five-pound or more weight gain or worsening foot swelling (pedal edema) should be reported to the prescriber to prevent acute pulmonary edema
and hospital admission for CHF.