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(myocardosis), a term often applied to a broad group of heart diseases; specifically, noninflammatory lesions of the heart muscle (myocardium) resulting from a disturbance in myocardial metabolism. Among the causes of cardiomyopathy are nutritional disorders (alimentary dystrophy and avitaminosis, for example); protein metabolism disorders in hepatic or renal insufficiency and podagra; disturbances of carbohydrate metabolism (diabetes mellitus) and electrolyte metabolism; and endocrine disorders such as those associated with thyrotoxicosis and with hypoxia in impairment of coronary circulation, anemia, and mountain sickness. Myocardosis may also be caused by overstraining the myocardium and by exogenous poisons, such as carbon monoxide and alcohol.

In many cases the patient has no specific symptoms in the early stages; however, there may be shortness of breath and disagreeable sensations in the heart. Cardiomyopathy is manifested by dull, distant heart sounds, electrocardiographic changes, systolic murmur, extrasystole, and, more rarely, other types of arrhythmia. Severe cardiomyopathy weakens the heart contractions and may cause cardiac insufficiency. The changes associated with cardiomyopathy are usually reversible and disappear with the elimination of the underlying disease.

The cure includes treatment of the underlying disease and administration of agents that improve metabolic processes in the myocardium.


Kedrov, A. A. Bolezni myshtsy serdtsa. Leningrad, 1963.


References in periodicals archive ?
2005), and research with dogs has shown that oral nicotine administration increases the degree of scarring that accompanies alcoholic cardiomyopathy (Rajiyah et al.
Long-term heavy drinking can cause the heart to become enlarged and lose some of its ability to contract, a condition known as alcoholic cardiomyopathy.
binge) and long-term drinking can interfere with the function of the heart, a condition referred to as alcoholic cardiomyopathy.
The, article then summarizes the results of studies aimed at identifying the mechanisms underlying alcohol-induced liver damage, alcoholic cardiomyopathy, and FAS.
The most convincing circumstantial evidence for alcoholic cardiomyopathy is the extensive data, in animals and humans, of nonspecific cardiac abnormalities related to alcohol.