allergic reaction

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allergic reaction

[ə′lərj·ik rē′ak·shən]
(medicine)
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
Platelet P-selectin is required for pulmonary eosinophil and lymphocyte recruitment in a murine model of allergic inflammation. Blood 2005;105:2074-81.
Clinical-stage company Connect Biopharmaceuticals Ltd said on Monday that it has filed an application in Australia for the planned phase 1 clinical trial of its monoclonal antibody candidate CBP-201 for allergic inflammation.
The lack of Tregs causes the emergence of allergic inflammation along with the increase in Th2 cells [4].
The key cytokines responsible for the allergic response include interleukin- (IL-) 4, IL-5, and IL-13, as well as innate lymphoid (ILC-2) cells which may amplify and maintain local Th2-driven allergic inflammation by secreting Th2 cytokines, particularly IL-5 and IL-13 [55].
The pathophysiological roles of PI3Ks and therapeutic potential of selective inhibitors in allergic inflammation. Int Arch Allergy Immunol 2010; 152: 90-95, doi: 10.1159/ 000312132.
Although protein expression for IL-22 BP remains to prove, our preliminary study suggests that IL-22 BP may play a role in a Th2 cytokine driven allergic inflammation as natural antagonist for IL-22.
In light of recent developments surrounding tumor immunotherapy, the researchers decided to combine 5-FU with calcipotriol (calcipotriene, approved for psoriasis), which induces thymic stromal lymphopoietin (TSLP), "an epithelium-derived cytokine and a master regulator of allergic inflammation in the skin," according to the authors.
have previously demonstrated the significance of pendrin in the pathogenesis of airway allergic inflammation [7, 8].
It was confirmed that functional TLR4 agonists can induce TSLP expression to trigger TH2-mediated allergic inflammation through TLR4-dependent innate immune pathways [16].
Specifically, among epithelium-associated cytokines, IL-25 exacerbates allergic inflammation by epithelial cell hyperplasia, mucus secretion, airway hyperresponsiveness, and production of specific Th2 cytokines [28,29].
Molecular mechanisms of cytokine and chemokine release from eosinophils activated by IL-17A, IL-17F, and IL-23: implication for Th17 lymphocytes-mediated allergic inflammation. J Immunol.