autoantibody

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autoantibody

[¦ȯd·ō′ant·i‚bäd·ē]
(immunology)
An antibody formed by an individual against his own tissues; common in hemolytic anemias.
References in periodicals archive ?
Frequency of Diabetes and Thyroid Autoantibodies in Patients with Type 1 Diabetes and Their Siblings.
Some autoantibodies that are identified as diagnostic biomarkers may be protective and have potential in themselves as therapeutic biomolecules.
For these cases, corticosteroid treatment is administered to stop the development of autoantibodies (2).
The new criteria emphasized the purpose of anti-SRP and anti-HMGCR autoantibodies. If both autoantibodies are negative, then clinical (proximal muscle weakness and high creatine kinase) and pathological requirements (necrotic fibers, myophagocytosis, regeneration, and paucilymphocytic infiltrate) need to be satisfied.
The methodology used in this study is capable of detecting autoantibodies against all three of these ZnT8 variants ZnT8A variant distribution in Turkish T1D children could not be determined.
In an article (21), it has been informed that anti-mitotic apparatus antibody is one of the rare autoantibodies in patients who have symptoms related to autoimmunity.
With reference to joint manifestations seen among patients with JIIM and anti-MDA5 autoantibodies, the pathology and prognosis, including the response to medical treatments for myositis, have not been well described.
Circulating autoantibodies to whole myosin molecules were detected in 26%-46% of the patients who had myocarditis or dilated cardiomyopathy; in these patients, the autoimmune reactivity did not show significant difference between [alpha]-myosin and [beta]-myosin [124-126].
In some of the other hypersensitivity conditions, there is a female predominance, an increased frequency of HT-related autoantibodies and/or positive ANA, and autoimmune mechanisms are also thought to play a role in a subgroup of patients.
ANA autoantibodies, C3c and renal damage between gluten and standard diet group was non-significant.
We strongly suspected neutralizing anti-IFN-[gamma] autoantibodies as the cause because the whole blood IFN-[gamma] level with mitogen stimulation was low, as determined using an IFN-[gamma]-releasing assay (QuantiFERON TB 3G; Cellestis, Carnagie, VIC, Australia).
Various studies have reported that autoantibodies to CA isoenzymes increase during the course of some immunological diseases.