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Related to CXCL9: CXCL10, CXCL11
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Chemokines whose concentrations were measured in the present study included CXCL8 (IL-8), CXCL9 (monokine induced by gamma interferon), CXCL10 (interferon-gamma-induced protein 10 [IP-10]), CXCL11 (interferon-inducible T-cell alpha chemoattractant), CCL4 (macrophage inflammatory protein 1[sz] [MIP-1[sz]]), CCL11 (eotaxin-1), CCL26 (eotaxin-3), CCL17 (thymus and activation-regulated chemokine [TARC]), CCL22 (macrophage-derived chemokine [MDC]), CCL2 (monocyte chemoattractant protein 1 [MCP-1]), and CCL13 (MCP-4).
10) Thus, NOD1 activation of CXCL9, CXCL10, and CXCL11 transcription via type I IFN requires nuclear translocation of ISGF3, a complex composed of Stat1, Stat2, and IRF9.
BAL (A) IL-5, (B) IL-13, (C) IL-9, (D) CXCL1, (f) IL-6, (F) IL-2, (G) eotaxin (CCL11), (H) CSF3, (I) IL-1a, (J) IL-10, (K IL-12 (p40), (L) CXCL10, (M) LIF, (N) RANTES, (O) CXCL9, and (P) CCL4 in a cohort of WT and db/db treated with isotype antibody prior to air or [O.
Mouse CCL4, CXCL9, and CXCL10 proteins were produced in yeast without the use of tags.
Increases in urine levels of the immune protein, called CXCL9, often show up in patients a month before an episode of organ rejection, researchers report August 22 in the American Journal of Transplantation.
Relationship between circulating levels of IFN-gamma, IL-10, CXCL9 and CCL2 in pulmonary and extrapulmonary tuberculosis is dependent on disease severity.
La coexpresion de CXCL9 y CXCL12 es un fuerte estimulo quimiotactico para los linfocitos infiltrantes de tumores CXCR4+/CXCR3+/CD8+ y para los linfocitos B malignos CXCR4 + /CXCR3-, en el microambiente perivascular; tambien, hay un aumento del factor de crecimiento de celulas B: interleuquina 4 en el LPSNC, asi como sobreexpresion de los protooncogenes c-myc y Pim-1 (8).
DNA microarray analysis of several genes known to be strongly regulated during gastrointestinal inflammation, combined with quantitative real-time PCR (qRT-PCR) revealed that the apple juice extract AE04 (100-200 microg/mL) significantly inhibited the expression of NF-kappaB regulated proinflammatory genes (TNF-alpha, IL-1beta, CXCL9, CXCL10), inflammatory relevant enzymes (COX-2, CYP3A4), and transcription factors (STAT1, IRF1) in LPS/IFN-gamma stimulated MonoMac6 cells without significant effects on the expression of house-keeping genes.
Analysis of CXCL9 and CXCR3 expression in a case of intravascular large B-cell lymphoma.
Serum levels of sTNF-R1, sTNF-R2 and CXCL9 correlate with disease activity in adult type paracoccidioidomycosis.
Since CXCR3 shares multiple ligands (CXCL4, CXCL9, CXCL10 and CXCL11), the goal of this study was to determine the key role of CXCR3 ligands associated with fatal CM in order to facilitate their development as biomarkerfs) of CM severity.
TLR ligands and cytokines induce CXCR3 ligands in endothelial cells: enhanced CXCL9 in autoimmune arthritis.