cardiac muscle

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cardiac muscle

[′kärd·ē‚ak ¦məs·əl]
(histology)
The principal tissue of the vertebrate heart; composed of a syncytium of striated muscle fibers.
References in periodicals archive ?
The purity of cardiomyocytes extracted using the Percoll density gradient centrifugation was determined by phenotypically characterizing the cells by immuno-cytochemical analysis with antibodies against sarcomeric tropomyosin (Sigma, USA), which is a cardiomyocyte marker, and vimentin (Sigma, USA), which is a fibroblast marker, as negative control.
Other researchers found that cardiomyocytes (cardiac muscle cells) in the heart and in the blood vessels of rats matured according to the same clock, despite being distant from each other in the body.
In 2013, Da-Zhi Wang, a cardiology researcher, identified a family of microRNAs called miR-17-92 that regulates the proliferation of cardiomyocytes.
After SI treatment, LDH release increased significantly in models 1, 2, 3, 4, and 5 compared with the C group ( P < 0.05), indicating that SI treatment produces severe cardiomyocyte injury [Figure 2].
To assess the reproducibility of inherent cardiomyocyte characteristics, four donors (ID#s 1083, 1134, 1308, and 1434) were selected to represent a wide range of baseline beat rates.
Taken together, these results highlight the biocompatibility, suitability and ease of using keratin coated surfaces for neonatal murine cardiomyocyte culture to study cardiac hypertrophy in vitro (6) and also the emphasize the importance of topography in assessing cardiac function.
miRNA-590, a miRNA that can induce adult cardiomyocyte proliferation, was recently shown to be able to replace HAND2 and MYOCD in GMT direct reprogramming experiments using human and porcine fibroblasts [38, 39].
The Hippo-YAP Signaling in Cardiomyocyte Hypertrophy and Apoptosis.
How to use and interpret the time line of cardiomyocyte progeny with essential cell lines of a beating myocardium?
To our knowledge, this is the first study which provided direct proof that BDNF is expressed in cardiomyocyte and that cardiomyocytes BDNF is reduced after sepsis.
Cumulative evidences suggest that ROS, implicated in reperfusion toxicity, can trigger cardiomyocyte apoptosis via the mitochondrial apoptosis pathway [24, 25].
Apoptosis is the major form of cardiomyocyte death in the failing heart.