pneumoconiosis

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Related to Coal workers' pneumoconiosis: black lung disease

pneumoconiosis

(no͞o'məkō'nēō`sĭs), chronic disease of the lungs. Primarily an occupational disease of miners, sandblasters, and metal grinders, it is a result of repeated inhalation of dusts, including iron oxides (e.g., rust and filings), silicates (e.g., talc and rock dust), and carbonates (especially coal dust). Particles collect in the lungs and become sites for the formation of fibrous nodules. As the disease progresses, fibrous tissue increasingly replaces elastic lung tissue. Loss of lung function is signaled by shortness of breath, wheezing, coughing, and difficulty in expectorating. A heart deficiency called cor pulmonale may develop in severe cases. Sufferers are particularly vulnerable to infectious lung diseases such as tuberculosis. Pneumoconiosis is incurable and treatment is purely symptomatic. Because the inhaled dusts cause darkening of the lung tissue, the disease is also known as black lung. Silicosis, the form of the disease prevalent among miners, is commonly called miner's lung.

pneumoconiosis

[¦nü·mō‚kō·nē′ō·səs]
(medicine)
Any lung disease caused by dust inhalation.
References in periodicals archive ?
Presence of stable coal radicals in autopsied coal miners' lungs and its possible correlation to coal workers' pneumoconiosis.
The CD exposure can cause coal workers' pneumoconiosis, a disease of coal miners characterized by the aggregation of dust-laden macrophages near the respiratory bronchioles to form structures known as macules.
Black lung disease, or coal workers' pneumoconiosis, is caused by inhaling dust produced during coal mining.
Of particular concern to us is the recent increase in prevalence of all forms of coal workers' pneumoconiosis, including progressive massive fibrosis.
Based on the first National Study of Coal Workers' Pneumoconiosis (CWP) and the U.