Zog

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Zog

(zŭk), 1895–1961, king of Albania. Originally Ahmet Muhtar Bej Zogolliu (later Albanianized to Zogu), he came from a Muslim family and served in the Austrian army in World War I. He became Albanian minister of the interior in 1920, minister of war in 1921, and premier in 1922. A revolution in 1924 led to his flight, but he returned with Yugoslav backing and became (Jan., 1925) president of the Albanian republic. Zog turned to Italy for financial aid. The Treaty of Tirana (1926) gave Italian loans in return for Albanian concessions; a defensive military alliance followed one year later. In 1928, Zog was proclaimed king as Zog I.

Albania's economy advanced during his reign, and a modern legal system was introduced. Zog attempted to avoid further Italian encroachment, but the appearance (1934) of an Italian fleet at Durazzo forced him into submission. In Apr., 1939, Italy invaded and quickly subdued Albania. Zog, who had married the Hungarian-American countess Geraldine Apponyi in 1938, fled with his queen and two-day-old son. Victor Emmanuel IIIVictor Emmanuel III,
1869–1947, king of Italy (1900–1946), emperor of Ethiopia (1936–43), king of Albania (1939–43), son and successor of Humbert I. In 1896 he married Princess Helena of Montenegro.
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, of Italy, was proclaimed king; he abdicated in 1943, and Communist partisans gained control of Albania (1944) and proclaimed it a people's republic (1946). Zog was in exile in England, Egypt, and France until his death; his remains were reinterred in Albania in 2012. Upon Zog's death, his son (1939–2011) was proclaimed Leka I by Albanian exiles; he eventually returned to Albania and died there.

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References in periodicals archive ?
Importantly, the work showed that when these stem cells are activated, they send signals back to the mother cells of the tissue to control the number of cells produced, through a molecular gene called Dlk1. In the same study, the researchers also demonstrated that Dlk1 can enhance stem cell activation and tissue regeneration in a wound-healing model.
Hu's team found that the mesenchymal stem cells can be activated by a gene called Dlk1 and that the process also supports tissue regeneration in tooth healing.A
Their study showed that a gene called Dlk1 enhances stem cell activation and tissue regeneration in tooth healing.
There were numerous moderate-to-strong correlations in the expression levels of these Cd-associated genes, with the strongest positive correlations between H19 and IGF2 (rho = 0.81; p <0.0001) and the strongest inverse correlation between delta-like noncanonical notch ligand 1 (DLK1) and SGCE (rho = -0.63; p < 0.0001) (Figure 1C).
MiR-129-5p inhibits non-small cell lung cancer cell stemness and chemoresistance through targeting DLK1. Biochem Biophys Res Commun 2017;490:309-16.
Kim et al., "Exogenous administration of DLK1 ameliorates hepatic steatosis and regulates gluconeogenesis via activation of AMPK," International Journal of Obesity, vol.
Livers of mice overexpressing the IGF2BP2 splice variant p62 highly expressed the stem cell marker DLK1 and secreted DLK1 into the blood; DLK1was previously shown to correspond with poor survival in HCC [27].
The miRNAs deregulated in GB and in neural precursor cells are clustered in seven genomic regions and have been associated with many other cancers like the mir-17 family cluster [57-59], miR-182-183 cluster [60], miR-302-367 and miR-372 [59, 61, 62], and the Dlk1 domain [63].
Wei et al., "MicroRNA-126-5p promotes endothelial proliferation and limits atherosclerosis by suppressing Dlk1," Nature Medicine, vol.
Apart from the influence on EC apoptosis, endothelial miR-126-5p could also promote the proliferation of ECs through suppression of the Notch1 inhibitor delta-like 1 homologue (Dlk1), thereby preventing the formation of atherosclerotic lesions [35].
The protein, DLK1, may provide the basis of a non-invasive pre-natal test, scientists believe.
Almost the same result was published by Micheal Georges' group analyzing the same imprinted region in the ovine and human genomes including DLK1, GTL2, PEG11, antiPEG11 and MEG8 in 2001.