enterocyte


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enterocyte

[′en·tə·rə‚sit]
(histology)
A cell that lines the intestinal wall.
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LPS crosses the enterocyte barrier coupled with lipoproteins, stimulating the innate immune system, TLR4 in adipocytes, and vascular tissue, activating NFkb, and increasing inflammation, oxidative stress, and immune dysfunction.
Feeding of oxidized fats to broilers and swine: Effects on enterocyte turnover, hepatocyte proliferation and the gut associated lymphoid tissue.
These models enable the determination of potential structural changes to the peptides that could maximize the ability to cross the enterocyte along with the permeation-enhancing system when formulated.
Such alterations characteristically lead to increased shedding of brush border membranes, accelerated cell-loss and shortened, sparse and irregular enterocyte microvilli, thus disrupting digestion and absorption.
We observed an intense, cytoplasmic, and granular labeling in enterocyte villi of inoculated birds only (Figure 1), suggesting a substantial intestinal replication of a TCoV-related virus.
H2AX is the phosphorylated histone protein breakdown product, which was noted both in the enterocyte nuclei as well as in the cytoplasm of control duodenal biopsy specimens.
Group D animals had significantly greater increase in enterocyte proliferation (indicating healing from damage) than animals in Group C.
Measurement of circulating mediators such as intestinal fatty acid binding protein (I-FABP) and citrulline may fill this gap, as they are validated indicators of enterocyte mass, villous atrophy and permeability.
The normally encoded protein regulates enterocyte export in response to hepcidin, while the mutant is resistant to internalisation and degradation, resulting in unregulated iron absorption.
Subadult abalone did not demonstrate the same response in the crop epithelium as juvenile abalone, and this could be a result of the enterocyte turnover rate, which could be masking the effects of epithelial hypertrophy in subadults compared with juveniles.
These glucosidases are comprised of two enterocyte membrane bond complexes of N- and C-terminals each, as well as of maltase-glucoamylase (MGAM) and sucrase-isomaltase, making them the ultimate step in producing dietary glucose.
Congenital enterocyte heparan sulphate deficiency with massive albumin loss, secretory diarrhoea, and malnutrition.