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Related to FUT2: Lewis Blood Group


symbol for the element seleniumselenium
, nonmetallic chemical element; symbol Se; at. no. 34; at. wt. 78.96; m.p. 217°C;; b.p. about 685°C;; sp. gr. 4.81 at 20°C;; valence −2, +4, or +6. Selenium is directly below sulfur in Group 16 of the periodic table.
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The country code for Sweden.


(1) (Special Edition) See iPhone SE.

(2) (Standard Edition) See Java 2.

(3) (Single Ended) The traditional type of signaling on the SCSI bus. See SCSI.

(4) See systems engineer and service engineer.

(5) (Second Edition) See Windows Second Edition.
References in periodicals archive ?
For 98 of these patients, blood samples were also collected at symptom onset for FUT2 genotyping by sequencing for the A385T and G428A nonsense mutations from total blood DNA (9).
6] Human genes: FUT2, fucosyltransferase 2 (secretor status included) (alias, galactoside 2-[alpha]-L-fucosyltransferase 2); FUT3, fucosyltransferase 3 (galactoside 3(4)-L-fucosyltransferase, Lewis blood group); MGAT5, mannosyl ([alpha]-1,6-)-glycoprotein [beta]-1,6-N-acetyl-glucosaminyltransferase.
In nearly all studies, people who lacked a functional copy of the FUT2 gene didn't get sick, even when deliberately exposed to this type of virus.
The para-Bombay blood group is caused by a deficient FUT1 (191), whereas the non-secretor blood group is caused by a deficient FUT2 (192).
A region of the FUT2 gene was amplified, by using the following primers: forward 5'-CCCATCTTCAGAATCACCCTGCCGGTGCTG-3' and reverse 5'-TCGGCCGGCCCGTGGAAACATCCCC AGGTA-3', which anneal at positions 280-309 and 535-564, respectively, as described (18).
a] structure is complex and involves at least three genes, FUT3, FUT2, and a sialyltransferase gene, that interact and compete with each other to produce the final products (Fig.
Although human NoV susceptibility is highly associated with secretor status, and thus with mutations in FUT2 (4), no information is yet available on whether host genetic factors determine susceptibility to SaV.