mushroom poisoning

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mushroom poisoning,

fungal poisoning caused by ingestion of certain mushrooms (fungal organisms), most commonly Amanita phalloides and Amanita muscaria and related species. Symptoms, caused by toxic peptides, may include severe abdominal pain, vomiting, cold sweat, diarrhea, and excessive thirst; they appear 8 to 12 hours after ingestion. Damage occurs largely in the liver and kidneys. Some mushrooms contain substances that produce hallucinatory states, e.g., Psilocybe mexicana (see hallucinogenic drughallucinogenic drug
, any of a group of substances that alter consciousness; also called psychotomimetic (i.e., mimicking psychosis), mind-expanding, or psychedelic drug.
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; ergotergot
, disease of rye and other cereals caused by the fungus Claviceps purpurea. The cottony, matlike body, or mycelium, of the fungus develops in the ovaries of the host plant; it eventually turns into a hard pink or purple body, the sclerotium, or ergot, that resembles
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). Occasional outbreaks of poisoning from eating canned mushrooms are not caused by poisonous mushrooms but by botulismbotulism
, acute poisoning resulting from ingestion of food containing toxins produced by the bacillus Clostridium botulinum. The bacterium can grow only in an anaerobic atmosphere, such as that found in canned foods.
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 resulting from improper canning methods.
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References in periodicals archive ?
It shows that GI lesions are common in these patients regardless of the presence or absence of GI symptoms. H.pylori infection was found in 24.7% patients (p<0.05).
For those patients given antiviral therapy for CMV viremia and immunosuppressive therapy for GI GVHD simultaneously, it is unclear whether GI symptom resolution was attributable solely to the immunosuppressive therapy (and thus, GI GVHD with CMV viremia but without CMV gut disease) or was due to both therapies in combination (and thus, GI GVHD with CMV gut disease).
During our analysis, we looked at the total amount of 32 different vitamins and minerals contained in the diets of the participants and how this related to the participant's GI symptoms. We found that total levels of the vitamins and minerals were not associated with GI symptoms in these children.
Little regional evidence is available about the approach to young patients with IDA who have GI symptoms. Considering that both GI symptoms and anemia are very common it is important to identify which patients genuinely need endoscopies.
In contrast, despite possible recall bias in our study, seroprevalence was higher for patients who reported fever and GI symptoms (i.e., SFTS symptoms) during the previous 3 years.
Similarly, in another trial, studying the effect of gluten challenge in NCGS patients, there was no observable difference in the GI symptoms of the control and test cohorts, once FODMAPs were removed from both gluten-free and gluten-containing flours [15].
Conflicting results have been reported concerning the association between upper GI symptoms, as measured by GCSI, in diabetes and objective measures of gastroparesis, e.g., scintigraphic measures of gastric emptying [44-46].
Out of 186 patients, 81 (43.54%) patients despite having upper GI symptoms had normal finding in the report.
Four patients had dyspnoea and one had upper GI symptoms. One patient had sudden cardiac arrest was opened in ICU through median sternotomy but could not be revived.
Comprehensive self-management: A nine-session, nurse-delivered, comprehensive self-management intervention that involved cognitive behavioral strategies, diet, relaxation, and education was found to significantly improve GI symptoms and quality of life compared with usual care.
GSRS utilises a Likert scale, depending on intensity and frequency of GI symptoms experienced over the past 3 months according to a semiquantitative score where 1 meant absent, 2 meant mild, 3 meant moderate, and 4 meant severe and interfering with daily activities.
Her mornings were characterized with feelings of panic in association with her GI symptoms, similar to our patient.