Ryan JF, Mazza ME, Pang K, Matus DQ, Baxevanis AD, Martindale MQ, Finnerty JR (2007) Pre-Bilaterian Origins of the Hox
Cluster and the Hox
Code: Evidence from the Sea Anemone, Nematostella vectensis.
(10) When inputs and infrastructure variables were included (kind of school, school stratification, number of teachers, school location, number of libraries, computer classrooms, laboratories, among others) following Hox
, none of them help explain the variation in the adjusted average achievement among schools (See Appendix 1).
qRT-PCR: quantitative real-time polymerase chain reaction; UVB: ultraviolet B; HOTAIR: HOX
antisense intergenic RNA.
For example, the formulation of a common question in the study of HOX
proteins "Given the high degree of sequence conservation of the HD between HOX
proteins and therefore conservation of the DNA binding sites recognized by these HDs, how do the HOX
proteins function to specifically direct specific segmental identities during development?" asserts that there is phenotypic specificity of TF function; a mechanism for specificity is assumed and sought at the outset.
Therefore, we reviewed the published literature for the role of HOX
genes during differentiation of three main types of SC, namely, (i) embryonic SCs, (ii) adult SCs (hematopoietic SCs, colonic SCs, and mesenchymal SCs), and (iii) induced pluripotent SCs.
These observations strongly indicate that deregulation of HOX
pathways is a dominant mechanism of leukemia transformation [10, 18].
activity mimics the teratogenic effects of excess retinoic acid signaling.
includes a critical set of genes that help control the embryo's development plan from head to toe.
As suggested by Hox
(2002, p.59), all interaction effects between two continuous variables are modeled by comparing the effects of the interaction on those in the 25th and 75th percentiles of the moderating variable.
The ICCs at care group level were calculated by dividing the variation at care group level by the total variation (Hox
Thus, our present study attempted to identify HOX
genes involved in LSCC pathogenesis through a HOX
gene expression profile analysis.
Each vertebra embryologically formed from combination of caudal half of one sclerotome and the cranial half of succeeding sclerotome regulated by HOX