Ménière's disease

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Related to Meniere's disease: vertigo, labyrinthitis

Ménière's disease

(mən-yĕrz`), disorder of the inner ear characterized by recurrent vertigovertigo
, sensations of moving in space or of objects moving about a person and the resultant difficulty in maintaining equilibrium. True vertigo, as distinguished from faintness, lightheadedness, and other forms of dizziness, occurs as a result of a disturbance of some part of
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 combined with hearing loss and tinnitustinnitus,
the hearing of sounds in the absence of any external sound, also known as ringing in the ears. The sounds may be perceived as hissing, whistling, buzzing, swooshing, roaring, or clicking in addition to ringing; in rare cases indistinct voices or music may be heard.
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 (a ringing sensation). It was first described by the French otologist Prosper Ménière, in 1861. The sufferer experiences severe dizziness, in which objects may seem to spin around, and often nausea, vomiting, and sweating. Attacks may last for several hours. In the disorder, which occurs most often in men between the ages of 40 and 60, the symptoms are the result of abnormally large amounts of a fluid (endolymph) collecting in the inner ear, but the exact cause of the disease or of the trigger for an attack is unclear. The disease is most typically treated by a reduced-sodium diet and diurectics; an acute attack may be treated by medications that control the vertigo and nausea. Vestibular rehabilitation therapy is sometimes used to help patient cope with the sense of imbalance that may occur between attacks. Surgical procedures that relieve the condition include vestibular neurectomy, labyrinthectomy, sacculotomy (placement of a stainless steel tack through the footplate of the stapes), ultrasonic irradiation, endolymphatic-subarachnoid shunt, and cryosurgery for relief of frequent vertiginous attacks and degenerative hearing.

Ménière’s Disease

 

a condition characterized by vertigo in combination with noise in one ear, impairment of hearing, nausea, vomiting, and loss of equilibrium; named after the French physician P. Ménière (1799-1862), who described it in 1861.

Ménière’s disease generally begins in patients between the ages of 25 and 40. Attacks may last for hours or even days. They are provoked by a decrease in the tonus of the vessels supplying blood to the peripheral portions of the vestibulocochlear nerve, which performs auditory and vestibular functions. According to another view, the disease is caused directly by increased fluid (endolymph) pressure in the inner ear. The vertigo is usually systemic in nature—that is, there is a sensation that surrounding objects or the patient himself is turning in a certain direction. Hearing is often unilaterally impaired after the attack passes. Loss of hearing progresses with repeated attacks, and deafness may result. Ménière-like conditions, with symptoms resembling Ménière’s syndrome, may also occur in some organic brain dis-eases (arachnoiditis, tumor of the cerebellopontile angle).

Ménière’s disease is treated with a series of strychnine and neostigmine-methylsulfate injections, with physical therapy (ultrahigh-frequency therapy, galvanic collar), and exercise. Drugs that dilate the blood vessels of the inner ear (pilocarpine) and brain (nicotinic acid, papaverine) and agents that decrease the excitability of the brain stem (atropine sulfate, chlorpromazine, haloperidol) are also prescribed. Surgery may sometimes be necessary.

REFERENCES

Velikov, K. A. Sindrom i boleznMen ’era. Moscow, 1967.
Patiakina, O. K., and T. D. Zadorova. “Simpozium o bolezni Men’era.”[New York, June 1965.] Vestnik otorinolaringologii, 1966, no. 5.
Bystrzanowska, T. Choroba Ménière’a. Warsaw, 1970.

V. A. KARLOV

References in periodicals archive ?
Patients were diagnosed with definite Meniere's disease according to the American Academy of Otolaryngology-Head and Neck Surgery (AAO-HNS) Committee on Hearing and Equilibrium criteria for Meniere's disease (1995).[sup][3] They met the diagnostic scale of at least two episodic vertiginous spells lasting 20 min or longer, a documented hearing loss in the affected ear at least once throughout progression of the disease, and subjective complaints of tinnitus or aural fullness.
Bautista Center) and ENT doctors have the equipment and expertise to properly diagnose Meniere's disease.
The diagnosis of Meniere's disease rests primarily on a meticulous history.
Comment: Meniere's disease (also called idiopathic endolymphatic hydrops) is characterized by an excess of fluid in the inner ear, which results in disturbances of balance and hearing.
This study is the first to compare patients with symptoms of Meniere's disease with a control population.
Subjects complaining of general dizziness, aged between 20 and 70 years, gave consent at the outpatient clinic and were then screened for inclusion criteria (meeting the American Academy of Otolaryngology-Head and Neck Surgery Committee on Hearing and Equilibrium criteria for probable Meniere's disease entailing a "washout period" of at least 5 days of any prior treatment before enrollment) [5] and exclusion criteria (illness of other systems that is not appropriate for manual treatment).
The proportion of correct diagnoses in our retrospective study is significantly lower than in other two prospective studys.18,19 It may be concluded that generalized VBI was thought to be verte-brobasilar artery TIA and infarction; vestibular and inner ear lesions (including benign paroxysmal positional vertigo, Meniere's disease, etc); Mental disorders associated with dizziness or vertigo (anxiety and depression) and other systemic diseases.
Objective: To determine the efficacy of vasodilator and diuretic therapies in the prophylaxis of Meniere's disease. A randomized controlled trial.
Meniere's disease relates to damage of the inner ear which causes changes in hearing and violent vertigo which can last from a few minutes to several hours.
The most poorly understood of inner ear disorders, Meniere's disease lacks an accepted model of pathophysiology, and the evaluation of treatments has been complicated by the high placebo response of the disorder and the disease's capricious clinical course.