completely occupied the lacunae and developing Havers's canals were seen (Fig.
RANKL: new insights into the control of bone remodeling.
This finding may be due to a negative feedback of the inflammatory mediators on the osteocyte
growth and multiplication.
PTH signaling in osteoprogenitors, instead of osteoblasts and osteocytes
, is indispensable for B cell differentiation .
number increased significantly in both the control and laser groups at the 8th week compared with the 4th week (p < 0.05).
Manolagas, "Inhibition of osteoblastogenesis and promotion of apoptosis of osteoblasts end osteocytes
by glucocorticoids potential mechanisms of their deleterious effects on bone," Journal of Clinical Investigation, vol.
The results of the present work are particularly noteworthy since relatively large differences in oxide thickness and surface roughness showed no differences in bone microarchitecture, ultrastructure, osteocyte
morphology, and the molecular composition of the interfacial tissue after 12 weeks of healing.
Osteonecrosis (ON), which involves the physiological remodeling of bone tissues , is thought to be caused by interruptions in blood flow, a decline in the function/number of osteoprogenitor cells, and apoptosis of the osteocytes
originated from the endosteal surface do not participate in forming repair tissues.
Histological osteonecrosis was determined as five contiguous empty osteocytic lacunae present in trabecular bone together with the loss of osteocytes
utilize GJIC to coordinate bone remodeling in response to anabolic factors and mechanical loading.
Bonewald, "The osteocyte
: an endocrine cell and more," Endocrine Reviews, vol.