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(1) (Personal Cell) An impending cellular transmission technology from Artemis Networks LLC, San Francisco, CA (www.artemis.com) that creates an independent 4G LTE channel for each user. Formerly called Distributed Input Distributed Output (DIDO), pCell implementation is compatible with standard LTE phones and tablets, and people can move between standard LTE cells and pCells. When in pCells, all users can obtain uninterrupted HD video streamed to their devices with five-bar signal strength.

Use the Interference
Employing software-defined radio (SDR) and Artemis-designed pWave transmitters, pCells exploit interference rather than trying to eliminate it. Radio signals are combined in real time to create a centimeter-sized cell for each mobile device. See software-defined radio and 4G.

(2) (PCell) (Parameterized CELL) A pre-designed circuit that is customized for each purpose when it is used.

(3) (P-Cell) (Parameterized CELL) A Korean cellular technology that divides a geographic area into a matrix for location detection.
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These studies also reported decreased insulin (glucose-stimulated and random circulating) concentrations following STZ treatment, which may be explained in part by reduced [beta]-cell mass, rather than solely impaired p-cell function.
These data suggest that p-cells in low fat-fed mice treated with STZ do not face increased demand for insulin secretion and thus can afford a deficit in [beta]-cell mass before any impairment in glycemic status ensues.
from China (manuscript titled "Circulating PGRN Levels Are Increased but Not Associated with Insulin Sensitivity or p-Cell Function in Chinese Obese Children") investigated the association of progranulin with insulin sensitivity or [beta]-cell function in a Chinese paediatric cohort, (ii) N.
However, it is unknown whether p-cell apoptosis is increased in offspring of diabetic rats.
2, Apoptotic p-cell was identified by TUNEL staining.
Zinman, "Short-term intensified insulin treatment in type 2 diabetes: long-term effects on P-cell function," Diabetes, Obesity & Metabolism, vol.
Holst, "Effect of 6-week course of glucagon-like peptide 1 on glycaemic control, insulin sensitivity, and P-cell function in type 2 diabetes: a parallel-group study," The Lancet, vol.
[22] Other mechanisms such as lipotoxicity [23] and inflammation [24] resulting owing to high calorie intake are also likely causes of decline in p-cell functioning.
Recent findings indicate that defects in P-cell function and increased insulin resistance occur early in the natural history of T2DM and can be present for 3 to 6 years before diagnosis.
The probable cause for hyperinsulinemia is increased basal insulin secretion and decreased hepatic insulin clearance.[9] In both non-obese and obese PCOS women, insulin secretion is inappropriately low for their degree of insulin resistance, suggesting the presence of pancreatic P-cell dysfunction in these patients.[10,11] Ehrmann and colleagues have demonstrated decreased postprandial insulin secretory responses as well as abnormalities in entrainment of insulin secretion to an oscillatory glucose infusion in PCOS women, consistent with P-cell dysfunction.[10] P-cell dysfunction can precede glucose intolerance in PCOS.[11]
It has been suggested that the best treatment, and a potential cure, for both type 1 mellitus (T1DM) and type 2 diabetes mellitus (T2DM), is to replace or regenerate the pancreatic p-cell mass [1].
Type 2 diabetes, though poorly understood, is also known to be a disease characterized by an inadequate p-cell response to the progressive insulin resistance that typically accompanies advancing age, inactivity, & weight gain.