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vasopressin (văzˌōprĕsˈĭn): see antidiuretic hormone.
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The following article is from The Great Soviet Encyclopedia (1979). It might be outdated or ideologically biased.



a hormone secreted by the posterior lobe of the hypophysis; causes constriction of vessels (acting on the smooth muscles of their walls) and increased blood pressure (pressor effect); also maintains at a proper level the reverse absorption of water in the straight tubules of the kidneys, that is, decreases the quantity of urine discharged (antidiuretic effect). Vasopressin is formed in the neurosecretory cells of the anterior nuclei of the hypothalmus, from which it enters the hypophysis along the nerve fibers. The antidiuretic action of the hormone is one of the factors that maintain the relative constancy of the water-salt metabolism in the bodies of vertebrate animals and man. A deficiency of vasopressin may lead to diabetes insipidus, in which discharge of urine is sharply increased. Vasopressin is contained in preparations obtained from the posterior hypophysis—Pituitrin and adiurekrin. Chemically, vasopressin is an octapeptide constructed of eight amino acids (in the majority of animals and in humans vasopressin consists of cysteine, tyrosine, phenylalanine, glutamine, asparagin, proline, arginine, and glycine; in pigs lysine is found instead of arginine). In structure and effect vasopressin is similar to another hormone of the hypophysis—oxytocin.


The Great Soviet Encyclopedia, 3rd Edition (1970-1979). © 2010 The Gale Group, Inc. All rights reserved.


A peptide hormone which is elaborated by the posterior pituitary and which has a pressor effect; used medicinally as an antidiuretic. Also known as antidiuretic hormone (ADH).
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
The following parameters were monitored before, during and after pitressin infusion (30 min): ST-segment elevation (height, mm) and duration (s); alteration of T-wave; duration (s) of the P-Q and Q-T intervals; and percentage incidence of the arrhythmias.
In all control guinea-pigs, intravenous administration of pitressin resulted in the typical electrocardiographic signs of coronary arterial spasm (ST-Segment elevation, increase voltage or inversion of the T-wave, prolongation of the P-Q and Q-T intervals), in an elevation of arterial blood pressure of 40.5[+ or -]3.0 mmHg and in the disturbances of cardiac rhythm characterized by ventricular extrasystoles followed by paroxysmal ventricular tachycardia of short duration, intervalated by bradicardic sinusal rhythm with atrioventricular blocks.
A pitressin infusion was titrated to decrease high urinary output of greater than 500cc per hour and lower serum osmolarity.