RPE


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RPE

(analytical chemistry)
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
In other words, RTI between high-intensity series prevents RPE from reaching critical values early, i.e., restores the ability of the central nervous system (CNS) to stimulate muscle work, thus keeping the athlete practicing physical exercise (Kay and collaborators, 2001; Marcora and collaborators, 2008; Minett e Duffield, 2014).
First, we used the reverse transcription polymerase chain reaction (RT-PCR) to examine RPE cell expression of receptors for mediators produced by mast cells, such as tryptase, histamine, TNF-[alpha], and various chemokines.
Previous studies have suggested that the RPE degeneration observed in dry AMD and gyrate atrophy is caused by various factors, including oxidative stress [5] and ornithine accumulation [6].
Modulation of extracellular matrix turnover by altered changes in the RPE secretion of MMPs may play an important role in the normal function and pathology of the retina [17], and because of its location, the RPE likely plays a role in local scleral growth regulation [18].
"In preparation for potential future commercialization, we also are introducing our new Thaw-and-Inject formulation, enabling rapid off-the-shelf administration of RPE cells upon thawing, which we believe will streamline the use of OpRegen by retinal surgeons," Culley said.
The RPE are regularly updated to improve and streamline the STL's procedures.
199A even if the counterparty is not an individual or an RPE. The commenter recommended that the exception be limited to scenarios in which the related party is an individual or an RPE and that the term "related party" be defined with reference to existing attribution rules under Sec.
Relationships between the cardiometabolic variables (HR and [lactate)] and RPE were established through Pearson's correlation and linear regression.
In this study, we compared the proliferative and cytotoxic effects of asbercept (0.5 mg/mL), bevacizumab (0.3125 mg/mL), and ranibizumab (0.125 mg/mL) on RPE cell cultures by evaluating viability, apoptosis, proliferation, and senescence in control and drug-treated cells after 72 hours.
Age-related macular degeneration (AMD) can cause severe central vision loss in patients aged 75 or older,[1] which is initially caused by an age-related, progressive degeneration in the retinal pigment epithelium in the macular area of the retina.[2],[3] RPE cells, located between the neuroretina and the choriocapillaris, which comprise the outer blood-retinal barrier, participate in the selective transport of metabolites, phagocytose the outer segments that are shed from photoreceptors, and act as antigen-presenting cells.[4],[5],[6] A literature review has demonstrated that photoreceptor outer segment phagocytosis, peroxidized lipid membranes, and photo-oxidative reactive oxygen intermediates are endogenous sources of oxidative stress, which can induce damage to the RPE cells.[3]
The aims of this study were to evaluate the migratory ability of ASCs under RPE-oxidative stress conditions, as well as the protective role of ASCs on RPE cell death and degeneration of surrounding tissues.
The association of RPE alterations with one type of glomerulonephritis was first reported in 1989 [4].