Renal Tubules


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Renal Tubules

 

(1) In some invertebrates, that part of the excretory organs, or nephridia, consisting of coiled tubules. (2) In vertebrates (including man), the renal tubules constitute a large part of the nephrons of the pronephroi, the mesonephros, and the metanephros. When ciliated funnels are present (in some adult Selachii and amphibians), the renal tubules open into the coelom. When there are no funnels (in the postembryonic period of all other vertebrates), they terminate blindly. At the opposite end, the renal tubules empty into the ureters through the collecting tubules.

References in periodicals archive ?
The kidneys of rats exposed to Ti[O.sub.2] NPs for 24 or 48 h showed renal histological and histochemical alterations included the glomeruli, renal tubules and intracellular tissues.
The renal tubules have specialised features that allow the reabsorption and secretion of solutes, and osmosis of water, as the urinary filtrate travels from the glomerulus to the collecting ducts and then out of the kidney.
In addition, generation of ROS by BPA is the key event involved in necrosis of renal tubules leading to functional disturbances19.
The renal tubules were characterized by clearly-visible empty lumens without pathological deposits and shaded with a brush border.
Additionally, renal tubules showed unstained degenerated cytoplasmic regions of the lining epithelium, while arterioles showed diffuse muscular media thickening with perivascular edema, fibrosis, and minimal interstitium lymphocytic infiltrate.
In the present study, renal tubules showed severe tubular degeneration and fragmented tubular basement membrane in group B animals.
(15,16) Additionally, other studies have suggested that lymphoplasmacytic infiltrates surrounding renal tubules are associated with renal tubular defects.
Fanconi Syndrome, as it turns out, occurs when there is inadequate reabsorption of these molecules in the renal tubules. There are various known causes, including congenital defects, exposure to toxins (especially heavy metals), Vitamin D deficiency and adverse drug reactions.
The SGLT2 transporter is the main site for glucose reabsorption in the proximal renal tubules (DeFronzo, Davidson & Del Prato (2012).
The transection of IR rats (Figure 3B) revealed the presence of highly swollen renal tubules with epithelial denudation of the basement membrane (glomerular hypertrophy) and few necrotic tubules.
A histopathological analysis reveals features of arteriolopathy (hyalinization of nephron afferent arterioles), band-like interstitial fibrosis, and ischaemic atrophy of the renal tubules. The detection of changes typical for the CsA toxicity in a renal biopsy specimen attests to a considerable advancement of the process and has a nature of chronic and progressing remodelling of the nephron and tubulointerstitial compartment [7].
The renal tubules showed diffuse mild to moderate increase in thickness of the tubular basement membranes (TBM).

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