ENPNewswire-August 30, 2019--Advicenne Announces First Patient Enrolled in US ARENA-2 Pivotal Phase III Clinical Trial Evaluating ADV7103 in distal Renal Tubular
CHF Solutions announced the publication of a study in Circulation Heart Failure highlighting the benefits of decongestion outweigh concerns from previous studies that showed a rise in creatinine levels or tubular injury markers.The study, titled, "Renal Effects of Intensive Volume Removal in Heart Failure Patients With Preexisting Worsening Renal Function" further analyzed data on renal tubular
injury biomarkers from a subset of 105 participants in the CARRESS-HF trial.
dysfunction was reported in 10-22% of HIV-positive patients receiving TDF using variable parameters and definitions.
Medical conditions such as hyperparathyroidism and renal tubular
Distal Renal Tubular
Acidosis dRTA is an orphan disease characterised by a failure in the renal excretion of the acids generated through metabolism.
Among the most common causes of salt wasting crisis are congenital adrenal hyperplasia(CAH), isolated aldosterone deficiency, and peripheral resistance to aldosterone.1 Pseudohypoaldosteronism (PHA) is a disease characterized by renal tubular
unresponsiveness to aldosterone.
Increased level of concentration is also observed often with dehydration, renal tubular
acidosis, and acute renal failure.
Distal renal tubular
acidosis was found in 6% children.
Cadmium that is bound to metallothionein once released into plasma will be filtered into renal tubular
Fluorofenidone (AKF-PD) is a novel nonpeptide small molecule compound, which could delay the fibrosis process of various organs such as kidney, liver, and heart, inhibit the renal fibrosis factor such as TGF-[sz]1, besides, can significantly reduce the proliferation and activity of fibroblasts., The mechanism of anti-fibrosis of AKF-PD is also associated with the inhibition of OS, by inhibiting the production of ROS in NADPH-dependent renal tubular
Arthrogryposis, renal tubular
dysfunction, cholestasis (ARC) syndrome was originally described in 1973 by Lutz-Richner and Landolt (1) severe growth retardation, ichthyosis, recurrentfebriledisease, plateletabnormalities, sensorineural hearing loss, hypotonia and corpus callosum dysgenesis were later included as further features of the syndrome (1,2).