acanthosis


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Related to acanthosis: parakeratosis

acanthosis

[a‚kan′thō·səs]
(medicine)
Any thickening of the prickle-cell layer of the epidermis; associated with many skin diseases.
References in periodicals archive ?
From these observations, it may be added that besides the size of amyloid deposition, prominent hyperkeratosis, acanthosis and papillomatosis, other features like increased basal layer pigmentation and pigment incontinence (both more frequent in macular amyloidosis) as well as papillomatosis and periappendageal mononuclear and neutrophilic dermal infiltrate (more frequent in lichen amyloidosis) can be used for distinction between the two histologically.
Glycogenic acanthosis (GA) is a nodular or plaque-like elevation of the esophageal squamous epithelium, with an unknown origin (1).
Acanthosis nigricans is the most common dermatologic manifestation of obesity.
Histopathological findings acanthosis psoriaseforme, parakeratosis, and neutrophils accumulation in the stratum corneum (Munro microabscesses).
Based on skin lesions, their distribution and clinical signs, a diagnosis of acanthosis with a mixed skin lesions of Staphylococcus and Malassezia spp.
They comprised such characteristic features as lymphohistiocytic bandlike infiltrate occupying the upper dermis and obscuring the dermoepidermal junction, irregular acanthosis resembling the typical saw-toothed appearance, extensive liquefactive degeneration of the basal layer of the epidermis with subepidermal clefts (Max Joseph spaces), pigmentary incontinence, and numerous cytoid bodies forming huge clusters.
1,8,9) SSC can usually be differentiated from Candida esophagitis and glycogenic acanthosis, in which the plaques and nodules have discrete borders and are separated by normal intervening mucosa.
Significant trends toward smaller waist circumference and rate of acanthosis nigricans were seen with more vigorous activity.
Any other clinical conditions associated with insulin resistance (e g , severe obesity, Acanthosis nigricans); or
The hallmark of TCDD toxicity in humans is chloracne (Panteleyev and Bickers 2006), characterized by epidermal acanthosis and hyperkeratosis, and hyperkeratinization and metaplasia of the sebaceous glands, with comedone formation.