Schroeder, "Ablation of the liver fatty acid binding protein gene decreases fatty acyl CoA binding capacity and alters fatty acyl CoA pool distribution in mouse liver," Biochemistry, vol.
Kier, "Structural and functional characterization of a new recombinant histidine-tagged acyl CoA a binding protein (ACBP) from mouse," Protein Expression and Purification, vol.
The peroxisome proliferator (PP) response element (PPRE) upstream of the human acyl CoA oxidase gene is inactive in a sample human population: significance for species differences in response to PPs.
Species differences in response to peroxisome proliferators: increasing human PPAR alpha expression levels in human cells is not sufficient for fatty acyl CoA oxidase responsiveness.
cite the finding of an inactive PP response element upstream of the acyl CoA
oxidase gene in a sample human population (15) as additional evidence for why humans may be less responsive than rodents to PPs.
Diagnosis of medium chain acyl CoA
dehydrogenase deficiency by measurement of cis-4-decenoic acid in dried blood spots.
PPAR[Alpha]s from all of these species had similar binding affinities for rat acyl CoA oxidase (ACO) PPRE and comparable activities in reporter gene assays, indicating their functional similarity.
The peroxisome proliferator (PP) response element upstream of the human acyl CoA oxidase gene is inactive among a sample human population: significance for species differences in response to PPs.